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Published on: July 18, 2014
Constrictive pericarditis following heart transplantation: Reality or fiction?
Bahaa M Fadel1,2, Haifa Mahjoub1,3, Elias J Salem3
1King Faisal Specialist Hospital & Research Center, Riyadh, Saudi Arabia.
Insights
Constrictive pericarditis, a treatable cause of diastolic heart failure, can occur after heart transplantation. Early recognition is crucial in heart transplant recipients presenting with heart failure symptoms.
Area of Science:
- Cardiology
- Cardiac Surgery
- Transplantation Medicine
Background:
- Constrictive pericarditis (CP) is a significant cause of diastolic heart failure.
- Prior cardiac surgery is a known risk factor for developing CP.
- Heart transplantation is a complex procedure with potential long-term complications.
Observation:
- A case of constrictive pericarditis (CP) developing one year after heart transplantation is presented.
- Diagnostic delays in post-transplant CP can arise from misconceptions about pericardial presence in allografts.
- Factors like post-transplant effusion, mediastinitis, and rejection may increase CP risk.
Findings:
- Constrictive pericarditis can occur in heart transplant recipients.
- The absence of a native pericardium in transplanted hearts is a misconception that can delay diagnosis.
- Specific post-transplantation complications are associated with an increased risk of developing CP.
Implications:
- Clinicians should consider constrictive pericarditis in heart transplant recipients with unexplained heart failure.
- Awareness of this complication is vital for timely diagnosis and management.
- Further research may elucidate specific risk factors and diagnostic strategies for post-transplant CP.
Abstract:
Constrictive pericarditis (CP) is a curable cause of diastolic heart failure with prior cardiac surgery being a recognizable etiology. We report a patient who developed CP one year following heart transplantation. Several clinical and imaging related factors may lead to diagnostic delays in similar patients, including the mistaken belief that transplanted hearts are devoid of pericardium and thus do not develop constriction. Post-transplantation pericardial effusion, mediastinitis, and cardiac rejection predispose to future CP. Caretakers should consider this entity in allograft recipients who develop heart failure symptoms of unclear etiology.
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