Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Venous Thrombosis I: Introduction01:30

Venous Thrombosis I: Introduction

72
Venous thrombosis, the most common disorder of the veins, involves the formation of a thrombus or blood clot associated with vein inflammation. It can be classified as either superficial vein thrombosis or deep vein thrombosis.Superficial Vein Thrombosis: This involves the formation of a thrombus in a superficial vein, usually the greater or lesser saphenous vein. Though less severe than deep vein thrombosis (DVT), SVT can lead to complications if untreated.Deep Vein Thrombosis (DVT): This...
72
Venous Thrombosis III: Interprofessional Care01:29

Venous Thrombosis III: Interprofessional Care

57
Venous thrombosis requires effective prevention and treatment strategies to improve patient outcomes and reduce potential complications.Prevention StrategiesHealthcare providers must prioritize preventing venous thromboembolism (VTE) for all adult patients upon admission. Interventions depend on bleeding and thrombosis risk, medical history, current medications, diagnoses, planned procedures, and patient preferences. Patients on bed rest should change positions every two hours and, if not...
57
Venous Thrombosis II: Clinical Manifestations and Diagnostic Studies01:20

Venous Thrombosis II: Clinical Manifestations and Diagnostic Studies

66
The key difference between Superficial Vein Thrombosis (SVT) and Deep Vein Thrombosis (DVT) lies in their location and severity.Clinical ManifestationsSVT typically presents with localized pain, tenderness, and redness along the course of a superficial vein, often accompanied by a palpable, cord-like structure under the skin. This condition is usually less dangerous than DVT but can be uncomfortable and may lead to complications such as cellulitis or, rarely, a clot extension into the deep...
66
Anticoagulant Drugs: Low-Molecular-Weight Heparins01:30

Anticoagulant Drugs: Low-Molecular-Weight Heparins

1.0K
Hemostasis is a crucial process that prevents excessive blood loss from damaged blood vessels. It involves various mechanisms such as vasoconstriction, platelet adhesion and activation, and fibrin formation. The importance of each mechanism depends on the type of vessel injury. In contrast, thrombosis is the abnormal formation of a blood clot within the blood vessels, leading to potential complications if the clot obstructs blood flow. Thrombosis can be caused by increased coagulability of the...
1.0K
Pulmonary Embolism I: Introduction01:29

Pulmonary Embolism I: Introduction

177
Pulmonary embolism (PE) occurs when a thrombus, fat or air embolus, amniotic fluid, or tumor tissue blocks one or more pulmonary arteries. These blockages originate in the venous system or the right side of the heart.EtiologyPE primarily arises from deep vein thrombosis (DVT) and other hypercoagulable states, such as inherited thrombophilias. Additional etiological factors include venous stasis, commonly seen in obesity, and endothelial injury from surgery and trauma. Less common causes include...
177
Disorders of Hemostasis01:24

Disorders of Hemostasis

1.3K
Hemostasis, the process that stops bleeding after a blood vessel injury, is crucial for maintaining the integrity of the circulatory system. However, disorders of hemostasis can disrupt this delicate balance, leading to either excessive clotting or bleeding. These disorders can be broadly classified into thromboembolic disorders and bleeding disorders.
Thromboembolic Disorders
Two factors primarily cause thromboembolic conditions.
1.3K

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

A Rare Case of Multi-Organ Complications Due to Disseminated Citrobacter koseri Infection Originating From a Renal Abscess.

Cureus·2026
Same author

How Do We Remember the Future? A Systematic Review of ERP Components in Focal and Nonfocal Prospective Memory Tasks.

Biological psychology·2026
Same author

Probable Microcystin Toxicosis in a Red-Gartered Coot (<i>Fulica armillata</i>) from a Protected Coastal Wetland in Central Chile-A Sentinel for Toxic Cyanobacterial Bloom?

Veterinary sciences·2026
Same author

Implementing Digital Respiratory Technologies for People With Respiratory Conditions: Scoping Review.

Journal of medical Internet research·2026
Same author

Reversible podocytopathy in a patient with blastoid-variant mantle cell lymphoma: A case of paraneoplastic FSGS and a literature review.

Nefrologia·2026
Same author

A self-reliance framework for identifying strategic advanced materials.

Nature communications·2026

Related Experiment Video

Updated: Oct 12, 2025

In Vitro Microfluidic Disease Model to Study Whole Blood-Endothelial Interactions and Blood Clot Dynamics in Real-Time
09:19

In Vitro Microfluidic Disease Model to Study Whole Blood-Endothelial Interactions and Blood Clot Dynamics in Real-Time

Published on: May 24, 2020

9.2K

Thromboembolic Risk in COVID-19 Patients: Is There a Hidden Link?

José Cidade1, Hélder Pinheiro2, André Dias2

  • 1Internal Medicine, Centro Hospitalar Lisboa Ocidental - Hospital Egas Moniz, Lisboa, PRT.

Cureus
|November 22, 2021
PubMed
Summary

This study found no clear evidence of increased venous thromboembolism (VTE) risk in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pneumonia patients. D-dimer levels were not predictive of VTE events in this cohort.

Keywords:
covid-19d-dimerpulmonary embolismsars-cov-2venous thromboembolic disease

More Related Videos

Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models
04:40

Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models

Published on: January 5, 2024

2.7K
Author Spotlight: Deciphering Coagulation Disorders in Traumatic Brain Injury Patients
04:56

Author Spotlight: Deciphering Coagulation Disorders in Traumatic Brain Injury Patients

Published on: August 4, 2023

912

Related Experiment Videos

Last Updated: Oct 12, 2025

In Vitro Microfluidic Disease Model to Study Whole Blood-Endothelial Interactions and Blood Clot Dynamics in Real-Time
09:19

In Vitro Microfluidic Disease Model to Study Whole Blood-Endothelial Interactions and Blood Clot Dynamics in Real-Time

Published on: May 24, 2020

9.2K
Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models
04:40

Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models

Published on: January 5, 2024

2.7K
Author Spotlight: Deciphering Coagulation Disorders in Traumatic Brain Injury Patients
04:56

Author Spotlight: Deciphering Coagulation Disorders in Traumatic Brain Injury Patients

Published on: August 4, 2023

912

Area of Science:

  • Pulmonary Medicine
  • Hematology
  • Infectious Diseases

Background:

  • Emerging evidence suggests a high risk of venous thromboembolism (VTE) in patients with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pneumonia.
  • However, the actual incidence and optimal diagnostic strategies for VTE in these patients remain uncertain.

Purpose of the Study:

  • To determine the incidence of pulmonary embolism (PE) in patients with SARS-CoV-2 pneumonia.
  • To evaluate the role of D-dimer serum levels as a predictive factor for new VTE events in this population.

Main Methods:

  • A single-center retrospective observational cohort study was conducted.
  • Patients admitted with SARS-CoV-2 pneumonia and clinical/laboratory suspicion of VTE were included.
  • Statistical analysis included t-tests and Mann-Whitney U tests to compare groups.

Main Results:

  • The overall incidence of VTE was 30%, with thrombi predominantly found in segmental and subsegmental pulmonary artery branches.
  • No statistical difference in D-dimer serum levels was observed between patients with and without VTE, despite a tendency for higher values in the VTE group.
  • Mortality rates did not differ significantly between the groups.

Conclusions:

  • A definitive increased risk of VTE in SARS-CoV-2 pneumonia patients was not established.
  • The study did not demonstrate a link between VTE occurrence and a worse prognosis.
  • D-dimer serum levels are not recommended as predictors of VTE in SARS-CoV-2 pneumonia patients.