Influenza AM2 Channel Oligomerization Is Sensitive to Its Chemical Environment

Julia A Townsend1, Henry M Sanders1, Amber D Rolland2,3

  • 1Department of Chemistry and Biochemistry, The University of Arizona, Tucson, Arizona 85721, United States.

Analytical Chemistry
|November 23, 2021
PubMed

Insights

Influenza A matrix protein 2 (AM2) forms diverse oligomers, not just tetramers, influenced by its environment. This finding impacts understanding of influenza virus pathology and drug development.

Area of Science:

  • Virology
  • Structural Biology
  • Biophysics

Background:

  • Viroporins are viral ion channels crucial for infection and are established antiviral drug targets.
  • Influenza A matrix protein 2 (AM2) is a well-studied viroporin, previously thought to form only tetrameric structures.

Purpose of the Study:

  • To investigate the oligomeric state of the full-length and transmembrane (TM) domain of AM2.
  • To determine how environmental factors, including pH, detergents, and lipids, influence AM2 oligomerization.
  • To measure amantadine binding to AM2 within a lipid bilayer environment.

Main Methods:

  • Native mass spectrometry was employed to characterize AM2 oligomeric complexes.
  • AM2 was studied in various pH and detergent conditions, as well as within nanodiscs containing different lipids.
  • Amantadine binding stoichiometry was measured using nanodiscs.

Main Results:

  • AM2 forms a spectrum of oligomeric complexes, deviating from the established tetramer model.
  • Oligomeric states are significantly modulated by the local chemical environment, including lipids within nanodiscs.
  • Nanodiscs facilitated direct measurement of amantadine binding to AM2 in a native-like lipid bilayer.

Conclusions:

  • The oligomeric plasticity of AM2 is broader than previously understood, challenging existing paradigms.
  • Environmental factors critically influence AM2 structure and function, suggesting new avenues for antiviral strategies.
  • These findings offer novel insights into influenza virus pathology and potential drug development targets.