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Cellular uptake and cell-associated activity of third generation cephalosporins

Pediatric Research
|September 1, 1986
PubMed

Insights

Third-generation cephalosporins showed limited penetration into human polymorphonuclear leukocytes (PMNs), similar to penicillin. Antibiotic effectiveness against cell-associated Staphylococcus aureus and Haemophilus influenzae was multifactorial, not solely dependent on PMN penetration.

Area of Science:

  • Pharmacology
  • Microbiology
  • Immunology

Background:

  • Human polymorphonuclear leukocytes (PMNs) are crucial in combating bacterial infections.
  • Understanding antibiotic penetration into PMNs is vital for treating intracellular pathogens.
  • Third-generation cephalosporins are commonly used antibiotics with varying cellular activities.

Purpose of the Study:

  • To assess the penetration of third-generation cephalosporins into human PMNs.
  • To evaluate the antibacterial activity of these cephalosporins against cell-associated Staphylococcus aureus (SA) and Haemophilus influenzae type b (Hib).

Main Methods:

  • Radioactive uptake experiments were used to determine cellular to extracellular concentration ratios of cephalosporins in PMNs.
  • A preopsonized cell-associated bacterial assay with radiolabeled bacteria was employed to assess killing activity.
  • Antibiotic efficacy was compared between PMNs with antibiotics and PMN cell controls without antibiotics over time.

Main Results:

  • All studied third-generation cephalosporins exhibited low penetration into PMNs, with concentration ratios less than one.
  • Third-generation cephalosporins showed similar PMN exclusion patterns to penicillin.
  • Clindamycin demonstrated superior killing of SA, while nafcillin showed significantly less activity.
  • Chloramphenicol was more effective against cell-associated Hib than third-generation cephalosporins.
  • N-formimidoyl thienamycin required higher in vitro concentrations for good activity.

Conclusions:

  • Third-generation cephalosporins have poor penetration into human PMNs.
  • Antibacterial activity against cell-associated pathogens is influenced by multiple factors beyond cellular penetration.
  • Further research is needed to optimize antibiotic strategies for intracellular bacterial infections.

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