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Related Concept Videos

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Menopause, a natural biological process marking the end of a woman's fertility, typically occurs between the fifth and sixth decade of life. This phase is characterized by the exhaustion of the ovarian follicle pool, leading to less responsive ovaries despite the high levels of Follicle Stimulating Hormone (FSH) and Luteinizing Hormone (LH). The consequential decrease in estrogen production results in symptoms like hot flashes, heavy sweating, headaches, hair loss, muscle pains, vaginal...
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HIDRADENITIS SUPPURATIVA IN POSTMENOPAUSE.

C I Cucu1, C Giurcaneanu1,2, M M Mihai1,2

  • 1"Elias" University Emergency Hospital, Bucharest, Romania.

Acta Endocrinologica (Bucharest, Romania : 2005)
|December 20, 2021
PubMed
Summary

This case study highlights a rare instance of hidradenitis suppurativa (HS) with late-onset after menopause. It emphasizes the complex interplay of genetics, endocrine factors, and inflammation in HS development, particularly in older women.

Keywords:
androgenshidradenitis suppurativalate onsetmenopauseobesity

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Area of Science:

  • Dermatology
  • Endocrinology
  • Pathogenesis of Hidradenitis Suppurativa

Background:

  • Hidradenitis suppurativa (HS) is a chronic, inflammatory skin condition significantly impacting patient quality of life.
  • Late-onset HS, particularly postmenopausal, is uncommon and warrants further investigation into its specific triggers and mechanisms.

Observation:

  • A 68-year-old female presented with hidradenitis suppurativa (HS) 10 years after menopause, exhibiting symptoms like groin fistulas and vulvar anatomical changes.
  • The patient presented with metabolic syndrome features including obesity, hypertension, and dyslipidemia, but lacked signs of virilism or hormonal abnormalities.

Findings:

  • The case demonstrates a rare occurrence of HS with late onset, suggesting that factors beyond typical hormonal fluctuations may contribute to disease development.
  • Effective management involved antiseptics, topical retinoids, and antibiotics, indicating a multi-faceted treatment approach.

Implications:

  • This case underscores the complex pathogenesis of HS, involving genetic predisposition, endocrine dysregulation, metabolic alterations, and inflammation, even in postmenopausal women.
  • Further research into the precise role of endocrine factors and sex hormones in HS onset and progression is crucial for developing targeted therapies.