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Published on: October 16, 2021
Mitral Valve Repair for Barlow's Disease with Mitral Annular and Subvalvular Calcification: A Case Report
Kosuke Nakamae1, Takashi Oshitomi1, Kentaro Takaji1
1Division of Cardiovascular Surgery, Saiseikai Kumamoto Hospital, Kumamoto, Japan.
Insights
Mitral valve repair for Barlow's disease with extensive calcification is complex. This case demonstrates a successful individualized surgical strategy involving resection and artificial chordae implantation.
Area of Science:
- Cardiovascular Surgery
- Cardiac Valve Disease
- Mitral Valve Repair
Background:
- Barlow's disease, characterized by myxomatous degeneration of the mitral valve, can present with severe mitral annular calcification (MAC).
- MAC encasing the subvalvular apparatus (leaflets and chordae) poses significant challenges for mitral valve repair.
- Surgical repair in such complex cases requires tailored strategies to address both Barlow's disease and extensive calcification.
Observation:
- A 60-year-old woman presented with mitral valve regurgitation due to Barlow's disease and extensive MAC.
- The calcification involved the mitral valve leaflets and subvalvular apparatus, complicating standard repair techniques.
Findings:
- A successful mitral valve repair was achieved by resecting specific calcified areas (P2 rough zone, excess leaflet regions) while preserving other calcifications (P3).
- The procedure involved implanting artificial chordae and an annuloplasty ring to restore valve function.
- This individualized approach effectively controlled mitral valve regurgitation.
Implications:
- This case highlights the feasibility of complex mitral valve repair in rare instances of Barlow's disease with extensive MAC.
- An individualized and compounded surgical strategy is crucial for successful outcomes in these challenging cases.
- The findings suggest that meticulous surgical planning and technique can overcome significant anatomical obstacles in mitral valve surgery.
Abstract:
Barlow's disease with mitral annular calcification encompassing the subvalvular apparatus, including the valve leaflet and chordae, is extremely rare, and mitral valve repair in such cases is challenging. We report a case of a 60-year-old woman with mitral valve regurgitation that was successfully controlled by resecting the rough zone of P2 and calcifications on the excess leaflet regions and subvalvular apparatus, while retaining the calcification of P3 and implanting artificial chordae and an annuloplasty ring. Mitral valve repair for such cases requires an individualized and compounded surgical strategy for the technique to treat Barlow's disease and manage calcification to control mitral regurgitation.
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