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High altitude cerebral edema.

A J Hamilton, A Cymmerman, P M Black

    Neurosurgery
    |November 1, 1986
    PubMed
    Summary
    This summary is machine-generated.

    Acute mountain sickness (AMS) can progress to life-threatening high altitude cerebral edema (HACE). This review examines HACE epidemiology, clinical presentation, pathology, and treatment strategies for high-altitude environments.

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    Area of Science:

    • Neurology
    • Altitude Medicine
    • Physiology

    Background:

    • Acute mountain sickness (AMS) affects healthy individuals ascending rapidly to high altitudes without acclimatization.
    • Severe AMS can lead to life-threatening conditions like high altitude cerebral edema (HACE) and pulmonary edema.
    • HACE presents with diverse neurological symptoms and signs, offering insights into hypoxia's CNS effects.

    Observation:

    • HACE is a rare but severe complication of high-altitude exposure.
    • Neurological manifestations of HACE are varied, including both generalized and localized signs.
    • Climbers developing HACE provide a unique model for studying central nervous system responses to hypoxia.

    Findings:

    • This review synthesizes current knowledge on HACE epidemiology and clinical features.

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  • Pathological mechanisms and pathophysiological theories explaining HACE are examined.
  • Current recommendations for preventing and treating HACE are discussed.

    • Implications:

    • Understanding HACE is crucial for managing severe altitude illness.
    • Further research into HACE mechanisms can inform prevention and treatment strategies.
    • This review serves as a resource for clinicians and researchers in altitude medicine.