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Tarek Mohamed Abd El-Aziz1,2, Amanpreet Kaur3, Mark S Shapiro1
1Department of Cellular and Integrative Physiology, University of Texas Health Science Center at San Antonio, San Antonio, TX 78228, USA.
Phosphatidylinositol 4,5-bisphosphate (PIP2) binding sites on epithelial sodium channel (ENaC) subunits are crucial for channel activity. Mutations in these sites abolish ENaC
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