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Updated: Sep 27, 2025

Characterizing Mutational Load and Clonal Composition of Human Blood
Published on: July 11, 2019
An overview of mutational and copy number signatures in human cancer
Christopher D Steele1, Nischalan Pillay1,2, Ludmil B Alexandrov3,4,5
1Research Department of Pathology, Cancer Institute, University College London, London, UK.
Abstract:
The genome of each cell in the human body is constantly under assault from a plethora of exogenous and endogenous processes that can damage DNA. If not successfully repaired, DNA damage generally becomes permanently imprinted in cells, and all their progenies, as somatic mutations. In most cases, the patterns of these somatic mutations contain the tell-tale signs of the mutagenic processes that have imprinted and are termed mutational signatures. Recent pan-cancer genomic analyses have elucidated the compendium of mutational signatures for all types of small mutational events, including (1) single base substitutions, (2) doublet base substitutions, and (3) small insertions/deletions. In contrast to small mutational events, where, in most cases, DNA damage is a prerequisite, aneuploidy, which refers to the abnormal number of chromosomes in a cell, usually develops from mistakes during DNA replication. Such mistakes include DNA replication stress, mitotic errors caused by faulty microtubule dynamics, or cohesion defects that contribute to chromosomal breakage and can lead to copy number (CN) alterations (CNAs) or even to structural rearrangements. These aberrations also leave behind genomic scars which can be inferred from sequencing as CN signatures and rearrangement signatures. The analyses of mutational signatures of small mutational events have been extensively reviewed, so we will not comprehensively re-examine them here. Rather, our focus will be on summarising the existing knowledge for mutational signatures of CNAs. As studying CN signatures is an emerging field, we briefly summarise the utility that mutational signatures of small mutational events have provided in basic science, cancer treatment, and cancer prevention, and we emphasise the future role that CN signatures may play in each of these fields. © 2022 The Authors. The Journal of Pathology published by John Wiley & Sons Ltd on behalf of The Pathological Society of Great Britain and Ireland.
Insights
DNA damage creates somatic mutations with distinct patterns called mutational signatures. This review focuses on copy number alteration signatures, exploring their potential in cancer research and treatment.
Area of Science:
- Genomics
- Cancer Biology
- Molecular Pathology
Background:
- Cellular genomes face constant DNA damage from internal and external factors.
- Unrepaired DNA damage leads to somatic mutations, which carry signatures of mutagenic processes.
- While small mutational signatures are well-studied, copy number alteration (CNA) signatures are an emerging area.
Purpose of the Study:
- To summarize current knowledge on copy number alteration (CNA) signatures.
- To highlight the utility of small mutational signatures in basic science, cancer treatment, and prevention.
- To emphasize the future potential of CNA signatures in these fields.
Main Methods:
- Review of existing literature on mutational signatures.
- Focus on copy number alterations (CNAs) and their associated signatures.
- Analysis of genomic scars left by CNA events.
Main Results:
- Small mutational events (base substitutions, indels) have well-defined mutational signatures.
- Aneuploidy, leading to CNAs, arises from replication and mitotic errors.
- CNAs leave distinct genomic scars, termed copy number signatures.
Conclusions:
- Mutational signatures of small events are crucial in basic science, treatment, and prevention.
- Copy number signatures represent an emerging field with significant future potential.
- Understanding CNA signatures can advance cancer research, treatment, and prevention strategies.
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