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Alcoholic ketosis.

M Fulop, J Ben-Ezra, J Bock

    Alcoholism, Clinical and Experimental Research
    |December 1, 1986
    PubMed
    Summary
    This summary is machine-generated.

    Most patients with alcoholic ketosis stop drinking ethanol before admission and experience starvation. Starvation is a key factor in developing alcoholic ketosis, supported by low triiodothyronine levels.

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    Area of Science:

    • Biochemistry
    • Endocrinology
    • Gastroenterology

    Background:

    • Alcoholic ketosis is a metabolic complication associated with alcohol abuse.
    • The exact triggers and contributing factors, particularly the role of ethanol cessation and starvation, require further elucidation.

    Purpose of the Study:

    • To investigate objective evidence of ethanol cessation and starvation in patients with suspected alcoholic ketosis.
    • To identify the primary pathogenetic factors contributing to alcoholic ketosis.

    Main Methods:

    • Prospective study of 23 episodes of suspected alcoholic ketosis.
    • Measurement of plasma 3-hydroxybutyrate, ethanol, lactate, triiodothyronine, and insulin levels.
    • Assessment for evidence of ethanol consumption and starvation.

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    Main Results:

    • Most ketotic patients had no detectable blood ethanol.
    • Subnormal plasma triiodothyronine levels (<90 µg/dL) were observed in 6/7 ketotic patients, indicating starvation.
    • Ketotic patients typically presented with low-normal plasma insulin levels, consistent with starvation.

    Conclusions:

    • Findings support the hypothesis that most alcoholic ketosis patients cease ethanol consumption before admission.
    • Starvation is identified as a significant pathogenetic factor in alcoholic ketosis.
    • Lactic acidosis was predominant in non-ketotic patients.