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Related Concept Videos

Cancer02:18

Cancer

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Cancers arise due to mutations in genes involved in the regulation of cell division, which leads to unrestricted cell proliferation. Modern science and medicine have made great strides in the understanding and treatment of cancer, including eradicating cancer in some patients. However, there is still no cure for cancer. This is largely due to the fact that cancer is a large group of many diseases.
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Cancer Prevention02:59

Cancer Prevention

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Several factors can increase the risk of cancer in an individual. About 50% of cancer cases can be prevented by adopting a healthy lifestyle, regular exercise, eating healthy, and following a modest cancer prevention diet. Epidemiological studies have consistently shown that populations with vegetable and fruit-rich diets have reduced the incidence of cancer. On the other hand, populations who have a diet rich in animal fat, red meat, junk food, or high calories are predisposed to cancer.
Some...
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Mechanisms of Retrovirus-induced Cancers01:51

Mechanisms of Retrovirus-induced Cancers

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Retroviruses are RNA viruses that have been shown to cause cancers in diverse species, including chickens, mice, cats, and monkeys. The RNA genomes of these viruses are first reverse-transcribed into single and then double-stranded DNA (dsDNA) copies. This dsDNA called proviral DNA then integrates into the host genome. Subsequently, the host cell transcribes the proviral DNA in concert with the chromosomal DNA. This leads to the production of viral RNA and proteins that assemble at the host...
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Rous Sarcoma Virus (RSV) and Cancer01:03

Rous Sarcoma Virus (RSV) and Cancer

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Rous Sarcoma virus or RSV was discovered by F. Peyton Rous in the year 1911 as a filterable transmissible agent that could cause tumors in chickens. He won a Nobel Prize for this discovery in 1966. His experiments clearly demonstrated that some cancers could be caused by infectious agents and led to the discovery of many more cancer-causing viruses in animals as well as humans.
RSV is a retrovirus that contains two copies of a plus-strand  RNA genome. Its genome consists of four main open...
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Adaptive Mechanisms in Cancer Cells02:53

Adaptive Mechanisms in Cancer Cells

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Cancer cells accumulate genetic changes at an abnormally rapid rate due to the defects in the DNA repair mechanisms. From an evolutionary perspective, such genetic instability is advantageous for cancer development. Mutant cell lines accumulate a series of beneficial mutations that contribute to their progression into cancer.
Some of the advantages that cancer cells have on normal cells include - enhanced ability to divide without terminally differentiating, induce new blood vessel formation,...
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What is Cancer?02:12

What is Cancer?

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Cells and tissues must meticulously coordinate their activities for the normal functioning of the human body. Therefore, they exhibit socially responsible behavior - resting, growing, dividing, differentiating, or dying - for the organism’s benefit. Cancer arises when cells divide uncontrollably and invade other tissues or organs.
Although people have known about cancer for centuries, it was only in 1761 that Giovanni Morgagni of Padua performed a detailed autopsy of...
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Histopathological and molecular evaluation of hydatid cysts according to necropsy findings of 52 equids in Türkiye.

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Comparative Analysis of Nuclear (Ef1α) and Mitochondrial (mt-CO1 and mt-Nad5) Markers for Molecular Characterization of Sheep Isolates of Echinococcus granulosus sensu lato.

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Molecular evidence of <i>Macracanthorhynchus hirudinaceus</i> (Pallas, 1781) in cockchafers in rural areas of Elazig, Türkiye.

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First Histopathological and Molecular Characterization of Giant Thorny-headed Worm, Macracanthorhynchus hirudinaceus (Pallas, 1781) (Acanthocephala: Oligacanthorhynchidae) in Wild Boars, Sus scrofa Linnaeus, 1758 from Eastern Türkiye.

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Updated: Sep 22, 2025

In Vitro Modeling of Cancerous Neural Invasion: The Dorsal Root Ganglion Model
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In Vitro Modeling of Cancerous Neural Invasion: The Dorsal Root Ganglion Model

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Parasite and Cancer Relationship

Figen Çelik1, Sami Şimşek1

  • 1Fırat Üniversitesi Veteriner Fakültesi, Parazitoloji Anabilim Dalı, Elazığ, Türkiye

Turkiye Parazitolojii Dergisi
|May 23, 2022
PubMed
Summary

Parasites can influence cancer development, sometimes directly causing tumors or indirectly stimulating cancer growth. However, the immune response to certain parasites may also exhibit anti-cancer effects.

Area of Science:

  • Immunology
  • Oncology
  • Parasitology

Background:

  • Cancer and parasites share survival mechanisms, including resistance to apoptosis and immune evasion, making eradication difficult.
  • Parasites can directly cause cancer or indirectly promote its development through various mechanisms.
  • Immune responses to parasitic infections can exhibit anti-tumoral activity.

Purpose of the Study:

  • To explore the dual role of parasites in cancer development and potential anti-cancer effects.
  • To analyze the mechanisms by which parasites influence tumor growth and metastasis.
  • To investigate the immunomodulatory effects of parasites in the context of cancer.

Main Methods:

  • Review of in vitro and experimental animal studies on the parasite-cancer relationship.
Keywords:
Cancerparasitedrugantigen

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  • Analysis of mechanisms of parasite-induced carcinogenesis.
  • Examination of immune responses to parasites and their impact on tumor progression.

    • Main Results:

    • Parasites can directly cause cancer or indirectly stimulate tumor development.
    • Parasitic agents can modulate immune responses, affecting cancer progression.
    • Immune responses against parasites can demonstrate anti-tumoral activity, inhibiting metastasis and angiogenesis.

    Conclusions:

    • Parasites exhibit a complex, dual role in cancer, capable of both promoting and inhibiting tumor development.
    • Understanding the parasite-cancer relationship offers insights into novel immunotherapeutic strategies for cancer treatment.
    • Parasite-induced immune modulation presents a potential avenue for cancer prevention and therapy.