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Published on: March 7, 2019
Attachment Relationships as Determinants of Physical Health
Robert G Maunder1, Jonathan J Hunter1
1Associate Professor of Psychiatry, University of Toronto, and Mount Sinai Hospital, Toronto, Canada.
Insights
Childhood adversity can lead to insecure attachment, potentially harming physical health across a lifetime. This insecurity impacts stress responses, immunity, health behaviors, and substance use, increasing disease risk.
Area of Science:
- Psychology
- Developmental Psychology
- Health Psychology
Background:
- Childhood adversity disrupts secure attachment development.
- Attachment insecurity may negatively impact physical health throughout life.
Purpose of the Study:
- To review recent evidence linking attachment insecurity to physical health impairment.
- To explore proposed mechanisms through which attachment insecurity influences disease risk.
Main Methods:
- Literature review of recent evidence.
- Discussion of established and emerging mechanisms linking attachment and health.
Main Results:
- Attachment insecurity is associated with dysregulated physiological stress responses.
- Social relationship mediators link stress, immunity, and health.
- Relationship styles influence health behaviors and substance use.
- Substances like nicotine and alcohol can regulate negative affect, increasing disease risk.
Conclusions:
- Attachment insecurity presents a significant pathway to impaired physical health.
- Understanding these mechanisms is crucial for developing interventions.
- Further research is needed to fully elucidate the long-term health consequences of attachment insecurity.
Abstract:
Childhood adversity alters the relational world of the child and inhibits the development of secure attachment bonds. The purpose of this article is to survey recent evidence that attachment insecurity has the potential to impair physical health throughout the lifespan. It is proposed that attachment insecurity contributes to disease risk through a range of mechanisms which include (1) disturbances in arousal and recovery within physiological systems that respond to stress; (2) physiological links between the mediators of social relationships, stress, and immunity; (3) links between relationship style and various health behaviors; and (4) disease risk factors that serve as external regulators of dysphoric affect, such as nicotine and alcohol. The evidence for these mechanisms, particularly the evidence that has accumulated since the model was first proposed in 2000, is presented and discussed.
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