Proteomic alterations in extracellular vesicles induced by oncogenic PIK3CA mutations

Mayank Saraswat1,2,3, Kishore Garapati1,2,3, Jinyong Kim1

  • 1Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, USA.

Proteomics
|June 11, 2022
PubMed

Insights

Activating mutations in the PIK3CA gene alter the protein composition of extracellular vesicles (EVs). These changes include elevated cell adhesion molecules and markers of tumor invasion, impacting intercellular communication in cancer.

Area of Science:

  • Oncology
  • Molecular Biology
  • Cell Biology

Background:

  • PIK3CA is frequently mutated in human cancers, with E545K and H1047R being common activating mutations.
  • Intracellular signaling pathways affected by these mutations are known, but their impact on extracellular vesicles (EVs) is uncharacterized.

Purpose of the Study:

  • To investigate the proteomic changes in EVs derived from cells with PIK3CA activating mutations.
  • To understand how these mutations affect intercellular communication via EVs.

Main Methods:

  • MCF10A cells and isogenic mutants (PIK3CA E545K, H1047R) were cultured.
  • EVs were isolated using differential ultracentrifugation.
  • Proteomic analysis was performed using tandem mass tag (TMT) labeling and liquid chromatography tandem mass spectrometry (LC-MS/MS).

Main Results:

  • 579 proteins were identified in EVs, with 522 previously described.
  • EVs from both mutant cell lines showed elevated levels of cell adhesion molecules (Mesothelin, E-cadherin, EpCAM).
  • Markers associated with tumor invasion and progression (Galectin-3, TGFBI) were increased in mutant EVs.

Conclusions:

  • Activating PIK3CA mutations lead to significant alterations in EV protein composition.
  • These alterations include changes in cell adhesion molecules and tumor progression markers.
  • Mutant-derived EVs exhibit characteristic proteomic changes associated with these hotspot mutations.

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