CDK4/6 Inhibitors in Combination Therapies: Better in Company Than Alone: A Mini Review

Gian Luca Rampioni Vinciguerra1,2, Maura Sonego1, Ilenia Segatto1

  • 1Division of Molecular Oncology, Centro di Riferimento Oncologico di Aviano (CRO), Istituto di Ricovero e Cura a Carattere Scientifico (IRCCS), National Cancer Institute, Aviano, Italy.

Frontiers in Oncology
|June 17, 2022
PubMed

Insights

CDK4/6 inhibitors show promise for various cancers but face resistance. Research focuses on biomarkers and strategies to overcome both intrinsic and acquired resistance for improved therapeutic outcomes.

Area of Science:

  • Molecular Biology
  • Oncology
  • Pharmacology

Background:

  • Cyclin D-CDK4/6 complexes are crucial for cell cycle regulation.
  • Deregulation of this pathway drives cancer cell proliferation.
  • Selective CDK4/6 inhibitors are approved for hormone-positive advanced breast cancer.

Purpose of the Study:

  • To review predictive biomarkers for intrinsic resistance to CDK4/6 inhibitors.
  • To summarize therapeutic strategies for overcoming acquired resistance.
  • To explore expanding CDK4/6 inhibitor use beyond breast cancer.

Main Methods:

  • Review of clinical and preclinical studies on CDK4/6 inhibitors.
  • Analysis of identified resistance mechanisms (cell-cycle and non-cell-cycle related).
  • Focus on elucidating pathways for therapeutic targeting.

Main Results:

  • Approximately 30% of breast cancers exhibit intrinsic resistance to CDK4/6 inhibitors.
  • Acquired resistance develops in many patients with prolonged treatment.
  • Various adaptive strategies contribute to resistance, necessitating further research.

Conclusions:

  • Understanding resistance mechanisms is critical for effective CDK4/6 inhibitor therapy.
  • Targeting specific pathways may reverse resistant phenotypes.
  • Further research is needed to identify biomarkers and develop strategies to overcome resistance.

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