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Pulmonary microvascular changes during hyperdynamic sepsis in an ovine model.

D L Traber, G Schlag, H Redl

Circulatory Shock

|January 1, 1987

Summary

Sepsis induced by endotoxin initially reduces cardiac output and increases lung lymph flow, correlating with neutrophil activation and free radical formation. Later, increased cardiac output may help clear neutrophils, potentially reducing lung injury.

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Related Experiment Videos

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Related Concept Videos

Related Articles

Pre-clinical evaluation of liposomal gene transfer to improve dermal and epidermal regeneration.

[Pathophysiology of acute lung injury in severe burn and smoke inhalation injury].

Burn and smoke inhalation injury in sheep depletes vitamin E: kinetic studies using deuterated tocopherols.

[Treatment strategies for acute smoke inhalation injury].

Positive end-expiratory pressure ventilation increases extravascular lung water due to a decrease in lung lymph flow.

The role of the bronchial circulation in the acute lung injury resulting from burn and smoke inhalation.

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