Connecting residual depressive symptoms to self-reported executive functioning: A network analytical approach
Kristof Hoorelbeke1, Yannick Vander Zwalmen1, Bjørn Ingulfsvann Hagen2
1Psychopathology and Affective Neuroscience (PAN) lab, Department of Experimental Clinical and Health Psychology, Ghent University, Ghent, Belgium.
Persistent executive functioning (EF) impairments after depression remission are linked to specific symptoms. Depressive cognition strongly connects symptoms to EF issues like working memory and emotional control.
Area of Science:
- Neuroscience
- Psychiatry
- Psychology
Background:
- Persistent executive functioning (EF) impairments are a significant cause of disability after depression remission.
- Understanding the relationship between specific depressive symptoms and EF deficits is crucial for targeted interventions.
Purpose of the Study:
- To investigate the unique associations between cognitive, affective, and somatic depressive symptoms and self-reported EF in remitted depressed individuals.
- To explore how different dimensions of depression relate to specific EF domains using network analysis.
Main Methods:
- Network analysis was employed on data from 161 remitted depressed individuals.
- Self-reported EF was assessed using the Behavior Rating Inventory of Executive Function - Adult version (BRIEF-A).
- Depressive symptoms were measured using the Beck Depression Inventory 2nd edition (BDI-II).
Main Results:
- Three clusters emerged: Metacognition Index, Behavioral Regulation Index, and depressive symptomatology.
- Working memory and shifting difficulties acted as bridging nodes between EF and depressive symptoms.
- Depressive cognition most strongly linked depressive symptoms to EF clusters; all symptom dimensions related to emotional control impairments.
Conclusions:
- Depressive symptom dimensions show both shared and unique associations with EF domains.
- Specific EF difficulties, such as working memory, shifting, and planning, are linked to depressive symptomatology.
- Findings highlight the differential impact of depressive symptoms on EF, suggesting further research into EF's role post-depression remission.
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