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Updated: Aug 31, 2025

Reproducible Motor Deficit Following Aortic Occlusion in a Rat Model Of Spinal Cord Ischemia
Published on: July 22, 2017
Ischaemic lumbosacral plexopathy following aortic dissection.
Bruna Gutierres Gambirasio1, Rodrigo Matos Amaral1, Thiago Yoshinaga Tonholo Silva1
1Department of Neurology, Universidade Federal de Sao Paulo, Sao Paulo, Sao Paulo, Brazil.
A patient developed ischemic lumbosacral plexopathy after aortic dissection repair. This condition, affecting the nerves supplying the leg, resulted from reduced blood flow to the lumbar and lumbosacral plexus.
Area of Science:
- Cardiovascular Surgery
- Neurology
- Vascular Anatomy
Background:
- Acute myocardial infarction and Stanford type A aortic dissection pose significant surgical challenges.
- Vascular complications following aortic repair, such as limb ischemia, require prompt recognition and management.
Observation:
- A 57-year-old male presented with acute myocardial infarction and extensive aortic dissection.
- Post-Bentall procedure, the patient developed acute neurological deficits in the left leg, including weakness and sensory loss.
- Clinical examination revealed hypotonia, absent deep tendon reflexes, and significant motor deficits (MRC 0/5 distal, 3/5 proximal).
Findings:
- Electromyography confirmed subacute involvement of the left lumbar and lumbosacral plexus.
- MR imaging demonstrated diffuse muscle edema in the left gluteus maximus, consistent with ischemic injury.
- The diagnosis of ischemic lumbosacral plexopathy was established, secondary to aortic dissection and internal iliac artery occlusion.
Implications:
- This case highlights the potential for ischemic plexopathy as a complication of extensive aortic dissection and repair.
- Understanding the vascular anatomy of the lumbosacral plexus is crucial for diagnosing and managing such rare neurological sequelae.
- Early recognition and diagnostic workup, including electrodiagnostic studies and advanced imaging, are essential for appropriate patient care.
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