Atrial tachycardia related to atrial infarction treated with catheter ablation: a case report
Momo Fujiwara1, Soichiro Yamashita1, Makoto Takemoto1
1Hyogo Prefectural Awaji Medical Center, Hyogo Kenritsu Awaji Iryo Center, Shioya 1-1-137, Sumoto, Hyogo 6560021, Japan.
Insights
Atrial infarction, though under-recognized, can cause complex arrhythmias. Catheter ablation successfully treated atrial tachycardia in a patient with an infarcted atrial branch, demonstrating a novel therapeutic approach.
Area of Science:
- Cardiology
- Electrophysiology
- Interventional Cardiology
Background:
- Atrial infarction is often concurrent with ventricular infarction but remains under-recognized.
- Supraventricular tachyarrhythmias are common in atrial infarction, yet their underlying mechanisms are not fully understood.
Observation:
- A 51-year-old man presented with chest pain and ECG changes consistent with myocardial infarction.
- Coronary angiography revealed left circumflex artery occlusion, leading to jailed atrial branch occlusion post-stenting.
- The patient developed frequent premature atrial contractions progressing to atrial tachycardia resistant to medical management.
Findings:
- Electro-anatomical mapping identified a macro-reentrant atrial tachycardia circuit and premature atrial contractions originating from fragmented potentials in the right atrium posterior wall.
- These areas corresponded to the territory perfused by the occluded atrial branch of the left circumflex artery.
- Catheter ablation targeting the cavotricuspid isthmus terminated the tachycardia, and defragmentation of the right atrial posterior wall eliminated premature atrial contractions.
Implications:
- Electro-anatomical mapping is crucial for identifying tachycardia circuits and arrhythmia origins in atrial infarction.
- Atrial infarction can lead to complex arrhythmias due to multiple premature atrial contractions from infarcted myocardium.
- Catheter ablation offers a viable treatment for drug-resistant atrial tachycardia associated with atrial infarction.
Background:
Atrial infarction, usually concurrent with ventricular infarction, is under-recognized. Although most patients with atrial infarction have complicated supraventricular tachyarrhythmias, its mechanism is still unknown. We report a case of atrial tachycardia (AT) related to atrial infarction treated with catheter ablation.
Case Summary:
A 51-year-old man was referred for acute chest pain. Electrocardiography showed a junctional rhythm with ST depression in the precordial leads. Emergency coronary angiography revealed an occluded dominant left circumflex coronary artery (LCX). A drug-eluting stent was deployed; however, the atrial branch from the distal side of the LCX was jailed by the stent and became occluded. On the 7th day, the premature atrial contractions (PACs) became frequent and changed to AT. Owing to its resistance to medication, we performed catheter ablation. The electro-anatomical map revealed counter-clockwise macro-reentrant tachycardia at the tricuspid valve annulus, with low-voltage and fragmented potential (FP) areas at the posterior wall of the right atrium (RA). After terminating the AT through linear ablation for the cavotricuspid isthmus, multiple-focus PACs originating from the FP area in the RA posterior wall were documented. Coronary angiography revealed that these damaged areas were perfused by the atrial branch of the LCX. Defragmentation in the FP area could eliminate PACs. The patient was discharged with sinus rhythm and without any complications.
Discussion:
We can perform electro-anatomical mapping to identify tachycardia circuit and PACs arising from the FP area in the posterior RA, where the atrial branch was perfusing. Multiple PACs from infarcted myocardium result in tachycardia.
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