Unstable angina with coronary to pulmonary artery fistulae
Insights
Coronary artery fistulae, abnormal connections between coronary arteries and the pulmonary artery, were found in three patients with chest pain. These fistulae were not considered the cause of symptoms, though a coronary steal phenomenon was suggested in two cases.
Area of Science:
- Cardiology
- Cardiovascular Surgery
- Interventional Cardiology
Background:
- Coronary artery fistulae (CAF) are rare anomalies.
- Their presentation with chest pain can mimic other cardiac conditions.
- Understanding their clinical significance is crucial for diagnosis and management.
Observation:
- Three patients with chest pain underwent cardiac catheterization.
- All three were diagnosed with fistulae between the left anterior descending coronary artery and the main pulmonary artery.
- Two patients had concomitant coronary artery disease.
Findings:
- One patient with unstable angina and coronary artery disease was treated with percutaneous transluminal coronary angioplasty.
- Another patient with unstable angina underwent successful bypass grafting and fistula ligation.
- The third patient with atypical chest pain improved after reassurance, with the fistula deemed unrelated to symptoms.
Implications:
- Coronary artery fistulae may not always be the primary cause of chest pain.
- A coronary steal phenomenon is a potential contributing factor in patients with fistulae and coronary artery disease.
- Management strategies should consider the fistula's relationship to the patient's presenting complaint and underlying coronary health.
Abstract:
Three patients presenting with chest pain were found at cardiac catheterization to have fistulae between the left anterior descending coronary and the main pulmonary artery. Two patients presented with unstable angina, the third with atypical chest pain. Of the two patients presenting with unstable angina both had concomitant coronary artery disease. One was successfully treated with percutaneous transluminal coronary angioplasty. The other underwent successful double aorto-coronary bypass grafting and fistulae ligation. The remaining patient's symptoms resolved after informing him that he did not have atheromatous coronary artery disease. In no patient was the presence of the fistula felt to be related to the patients presenting complaint. In the two patients with unstable angina, a coronary steal phenomenon was postulated as a possible contributing factor.
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