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Related Concept Videos

Angle Closure Glaucoma: Treatment01:28

Angle Closure Glaucoma: Treatment

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Angle-closure glaucoma, or closed-angle glaucoma, is an eye condition where the iris bulges out and blocks the iridocorneal angle, resulting in a buildup of aqueous humor and increased intraocular pressure. Immediate medical attention is necessary due to the sudden onset of symptoms. The treatment for angle-closure glaucoma includes short-term and long-term approaches. Short-term treatment involves using eye drops like pilocarpine to lower intraocular pressure by increasing aqueous humor...
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Open Angle Glaucoma: Treatment01:27

Open Angle Glaucoma: Treatment

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In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
Drugs such as carbonic anhydrase inhibitors, α2- and...
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Glaucoma: Overview01:25

Glaucoma: Overview

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Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
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At the molecular level, visual signals trigger transformations in photopigment molecules, resulting in changes in the photoreceptor cell's membrane potential. The photon's energy level is denoted by its wavelength, with each specific wavelength of visible light associated with a distinct color. The spectral range of visible light, classified as electromagnetic radiation, spans from 380 to 720 nm. Electromagnetic radiation wavelengths exceeding 720 nm fall under the infrared category,...
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Updated: Aug 29, 2025

Full-Circle Cauterization of Limbal Vascular Plexus for Surgically Induced Glaucoma in Rodents
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[Drug-induced acute angle closure glaucoma].

M Masereel1, S Bonnet1, S Marchand1

  • 1Service d'Ophtalmologie, CHR Citadelle, Liège, Belgique.

Revue Medicale De Liege
|September 9, 2022
PubMed
Summary
This summary is machine-generated.

Acute angle closure glaucoma is an ophthalmic emergency. Recognizing symptoms like eye pain and blurred vision is crucial for urgent ophthalmologist treatment to prevent blindness.

Keywords:
MedicationRisk factorsAngle closure glaucoma

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Area of Science:

  • Ophthalmology
  • Medical Emergency

Background:

  • Acute angle closure glaucoma (AACG) is an ophthalmic emergency.
  • It can lead to irreversible blindness if not treated promptly.
  • Symptoms include ocular pain, blurred vision, headache, nausea, and vomiting.

Purpose of the Study:

  • To review the anatomical risk factors for AACG.
  • To identify drugs that may precipitate AACG in susceptible individuals.
  • To provide a brief overview of the pathophysiology of AACG.

Main Methods:

  • Literature review of anatomical risk factors.
  • Compilation of drugs known to induce AACG.
  • Summary of the pathophysiological mechanisms of AACG.

Main Results:

  • AACG presents with characteristic symptoms requiring urgent recognition.
  • Certain anatomical predispositions increase the risk of AACG.
  • Various medications can trigger AACG in at-risk patients.

Conclusions:

  • Prompt identification and management of AACG are essential to prevent vision loss.
  • Awareness of anatomical risk factors and drug-induced AACG is vital for clinicians.