Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Peripheral Arterial Disease II: Clinical Manifestations and Diagnostic Evaluation01:21

Peripheral Arterial Disease II: Clinical Manifestations and Diagnostic Evaluation

27
Clinical manifestationsPeripheral Arterial Disease (PAD) manifests through a range of symptoms, from the characteristic intermittent claudication to atypical presentations and severe complications in advanced stages. Intermittent claudication, a hallmark symptom of PAD, presents as exercise-induced muscle pain that typically resolves within minutes of rest. This pain is reproducible and stems from inadequate blood flow, leading to the accumulation of lactic acid produced during anaerobic...
27
Peripheral Artery Disease IV: Nursing Management01:26

Peripheral Artery Disease IV: Nursing Management

28
 The nursing management of a patient with peripheral artery disease (PAD) begins with a thorough assessment of the patient’s health history and clinical manifestations.AssessmentHealth History: Evaluate the patient’s history of hypertension, hyperlipidemia, family history of cardiovascular issues, and lifestyle factors such as dietary patterns, smoking, and physical activity.Physical Examination:Assess the affected extremity for decreased or absent peripheral pulses,...
28
Peripheral Artery Disease I: Introduction01:30

Peripheral Artery Disease I: Introduction

25
Peripheral artery disease (PAD) predominantly results from atherosclerosis, which involves the accumulation of fatty deposits, or plaques, within the walls of arteries. This causes them to narrow and harden, significantly reducing blood flow. PAD predominantly affects the legs, particularly the arteries supplying the thighs and calves. In rare cases, it may involve other arteries, including those in the arms.Etiology of PAD:The principal cause of PAD is atherosclerosis, which results from fatty...
25

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

BCG gone rogue: A case report of disseminated <i>Mycobacterium bovis</i>.

IDCases·2026
Same author

Yes, and: Leveraging peer-led professional development support as a bridge between the status quo and senior faculty-led efforts.

Journal of hospital medicine·2026
Same author

Forging physician-scientist professional identity: the roles of integration, relationships, and boundary crossing in training.

BMC medical education·2026
Same author

Unintentional ingestion of psychoactive cannabis products among adults: A scoping review.

The American journal of medicine·2026
Same author

Clinical Outcomes of Patients Managed in a Temporary COVID-19 Step-Down Unit.

Cureus·2026
Same author

Surgical hyoid bone repositioning effects on mandibular advancement and upper airway collapsibility: an anesthetized rabbit model study.

Frontiers in physiology·2025

Related Experiment Video

Updated: Aug 27, 2025

Positron Emission Tomography Using 64-Copper as a Tracer for the Study of Copper-Related Disorders
06:52

Positron Emission Tomography Using 64-Copper as a Tracer for the Study of Copper-Related Disorders

Published on: April 28, 2023

1.5K

Copper Deficiency and Polyneuropathy: A Case Report.

Hassan A Chami1, Mary Ann Kirkconnell Hall1

  • 1Department of Medicine, Division of Hospital Medicine, Emory University School of Medicine, Atlanta, USA.

Cureus
|September 26, 2022
PubMed
Summary

Copper deficiency can mimic other neurological conditions, presenting diagnostic challenges. Early recognition and supplementation are crucial to prevent permanent neurological damage, especially in patients with a history of bariatric surgery.

Keywords:
bariatric surgeryceruloplasmin transferrincopper deficiencycopper myeloneuropathypolyneuropathy

More Related Videos

Nerve Ultrasound Protocol to Detect Dysimmune Neuropathies
08:56

Nerve Ultrasound Protocol to Detect Dysimmune Neuropathies

Published on: October 7, 2021

2.8K
Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1
09:39

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1

Published on: February 13, 2018

9.6K

Related Experiment Videos

Last Updated: Aug 27, 2025

Positron Emission Tomography Using 64-Copper as a Tracer for the Study of Copper-Related Disorders
06:52

Positron Emission Tomography Using 64-Copper as a Tracer for the Study of Copper-Related Disorders

Published on: April 28, 2023

1.5K
Nerve Ultrasound Protocol to Detect Dysimmune Neuropathies
08:56

Nerve Ultrasound Protocol to Detect Dysimmune Neuropathies

Published on: October 7, 2021

2.8K
Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1
09:39

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1

Published on: February 13, 2018

9.6K

Area of Science:

  • Neurology
  • Nutritional Science

Background:

  • Copper is essential for physiological processes.
  • Copper deficiency symptoms can overlap with cobalamin deficiency (vitamin B12), complicating diagnosis.
  • Concurrent deficiencies of other micronutrients are common.

Observation:

  • A 50-year-old woman presented with altered mental status, limb weakness, numbness, and paresthesia.
  • Neurological examination revealed sensory neuropathy, ataxia, and pancytopenia.
  • Magnetic resonance imaging and cerebrospinal fluid analysis were normal.

Findings:

  • Nerve conduction studies indicated a generalized axonal sensorimotor polyneuropathy.
  • Copper deficiency was suspected due to a history of gastric bypass surgery.
  • Supplementation with copper was successfully initiated, leading to symptom improvement.

Implications:

  • Copper deficiency is increasingly recognized in hospitalized patients.
  • Delayed diagnosis can lead to irreversible neurological damage.
  • Physicians should consider copper deficiency in patients with polyneuropathy and pancytopenia, particularly after bariatric surgery.