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Negative Ulnar Variance Lessens DRUJ Instability After DRUJ Disruption: A Biomechanical Analysis
Hailey P Huddleston1, Joey S Kurtzman2, Liam Deegan3
1Hospital for Special Surgery, New York, NY, USA.
Ulnar variance (UV) impacts distal radioulnar joint (DRUJ) stability only after ligament and TFCC sectioning. Negative UV significantly reduced radial translation, suggesting a role in DRUJ instability management.
Area of Science:
- Orthopedic Surgery
- Biomechanics
- Anatomy
Background:
- Distal radioulnar joint (DRUJ) stability is crucial for forearm function.
- Ulnar variance (UV), the relative length of the ulna to the radius, is a known factor influencing DRUJ mechanics.
- Understanding UV's role in DRUJ stability is essential for treating wrist injuries.
Purpose of the Study:
- To biomechanically investigate the influence of ulnar variance (UV) on distal radioulnar joint (DRUJ) stability.
- To assess stability changes before and after sectioning of the DRUJ stabilizing structures.
Main Methods:
- Ten cadaveric forearm specimens were used.
- Baseline UV was measured, and radial translation was assessed using motion capture during a simulated Shuck test.
- A radial osteotomy was performed, and stability was re-evaluated at +4 mm, 0 mm, and -4 mm UV states before and after sectioning of the volar radioulnar ligaments and triangular fibrocartilage complex (TFCC).
Main Results:
- Sectioning of the volar radioulnar ligaments and TFCC significantly increased radial translation across all UV states.
- The ulnar negative (-4 mm) state demonstrated significantly lower radial translation compared to the positive (+4 mm) and neutral (0 mm) UV states after sectioning.
- No significant differences in translation were observed between UV groups when the DRUJ was intact.
Conclusions:
- Fixation at -4 mm ulnar negativity significantly decreased radial translation after sectioning of key stabilizing structures.
- Ulnar variance does not affect DRUJ stability when the joint is intact.
- Negative ulnar variance may play a protective role in DRUJ instability following ligamentous and TFCC disruption.
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