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[Parietal fibroplastic endocarditis].

J E Touze, D Charles, D Prigent

    Medecine Tropicale : Revue Du Corps De Sante Colonial
    |April 1, 1987
    PubMed
    Summary
    This summary is machine-generated.

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    Parietal fibroplastic endocarditis, linked to Löffler's and Davies' diseases, involves eosinophil granule proteins causing heart damage. Surgical interventions have significantly improved outcomes for affected children.

    Area of Science:

    • Cardiology
    • Pathology
    • Immunology

    Context:

    • Parietal fibroplastic endocarditis is associated with Löffler's endocarditis and Davies' endomyocardial fibrosis.
    • Damage results from cytolytic effects of eosinophil granule proteins, specifically major basic protein and cationic protein.
    • Lesion location (right, left, or both ventricles) dictates clinical manifestations, including valvular insufficiency and diastolic dysfunction.

    Purpose:

    • To elucidate the pathogenesis of parietal fibroplastic endocarditis.
    • To describe the clinical features based on lesion localization.
    • To highlight diagnostic modalities and recent therapeutic advancements.

    Summary:

    • Eosinophil granule proteins cause endocardial damage, with chronicity and eosinophil count influencing lesion severity.

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  • Clinical presentation varies with the ventricular involvement of endocarditis.
  • Two-dimensional echocardiography aids diagnosis, while ventricular angiocardiography is the gold standard.

    • Impact:

    • Surgical endocardiectomy and conservative valvular surgery have dramatically improved prognosis in pediatric cases.
    • Understanding the role of eosinophils provides targets for future therapies.
    • Improved diagnostic accuracy leads to earlier intervention and better patient outcomes.