Residual Interactions of LL-37 with POPC and POPE:POPG Bilayer Model Studied by All-Atom Molecular Dynamics

Muhammad Yusuf1, Wanda Destiarani2, Ade Rizqi Ridwan Firdaus2

  • 1Department of Chemistry, Faculty of Mathematics and Natural Sciences, Universitas Padjadjaran, Bandung 45363, Indonesia.

Insights

Antimicrobial peptide LL-37 interacts with bacterial membranes via specific residues, forming salt bridges. This interaction, particularly in residues 18-29, guides the design of more effective LL-37 derivatives.

Area of Science:

  • Biochemistry
  • Molecular Biology
  • Computational Chemistry

Background:

  • LL-37 is a potent antimicrobial peptide (AMP) with potential for bacterial membrane disruption.
  • Shorter LL-37 derivatives offer reduced costs and cytotoxicity, but require detailed interaction knowledge.
  • Previous simulations indicated LL-37 can penetrate lipid bilayers.

Purpose of the Study:

  • To elucidate the detailed residual interactions of LL-37 with bacterial (POPE:POPG) and eukaryotic (POPC) membrane models.
  • To identify key LL-37 residues involved in membrane interaction for derivative design.
  • To compare LL-37's interaction mechanisms with bacterial versus eukaryotic membranes.

Main Methods:

  • All-atom molecular dynamics simulations were employed.
  • Simulations utilized a simplified bacterial membrane model (POPE:POPG, 2:1) and a eukaryotic model (POPC).
  • Analysis focused on residue-level interactions and salt bridge formation.

Main Results:

  • Leucine-leucine residues were identified as the initial contact points with the membrane surface.
  • Cationic residues (Lys-Arg) formed salt bridges with POPE:POPG phosphate groups, facilitating penetration.
  • Eukaryotic POPC membranes showed shielded phosphate groups, hindering LL-37 penetration compared to bacterial membranes.
  • Residues 18-29 of LL-37 were identified as a core interaction region with the bacterial membrane model.

Conclusions:

  • LL-37's interaction with bacterial membranes is mediated by specific residues and salt bridge formation.
  • The core region (residues 18-29) is crucial for LL-37's selective interaction with bacterial membranes.
  • These findings provide a foundation for rationally designing optimized LL-37 derivatives with enhanced efficacy and reduced side effects.