Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

ECG Interpretation of Arrhythmias II: Atrial, Junctional and Ventricular Arrhythmias01:25

ECG Interpretation of Arrhythmias II: Atrial, Junctional and Ventricular Arrhythmias

79
Arrhythmia is a condition characterized by an irregular heart rhythm, with ECG changes that differ based on its origin and nature. The types of arrhythmias discussed below include atrial, junctional, and ventricular arrhythmias.Atrial ArrhythmiasPremature Atrial Complexes (PACs): PACs are early atrial beats caused by stress, caffeine, alcohol, electrolyte imbalances, hypoxia, hyperthyroidism, or certain medications (e.g., bronchodilators and decongestants). The ECG shows early P waves with an...
79
Disturbances in Heart Rhythm01:29

Disturbances in Heart Rhythm

1.1K
Arrhythmia or dysrhythmia refers to an abnormal heart rhythm caused by a defect in the heart's conduction system. It can cause the heart to beat irregularly, too quickly, or too slowly, leading to symptoms like chest pain, shortness of breath, and fainting. Factors such as stress, caffeine, alcohol, nicotine, cocaine, certain drugs, congenital defects, diseases, and electrolyte abnormalities can trigger arrhythmias.
Arrhythmias are categorized by their speed, rhythm, and origin. A slow heart...
1.1K
Coronary Artery Disease II: Pathophysiology01:26

Coronary Artery Disease II: Pathophysiology

23
Coronary Artery Disease (CAD) originates from a series of events that impair the function of coronary arteries, the blood vessels responsible for delivering oxygen-rich blood to the heart muscle. The pathophysiology of CAD is closely linked to atherosclerosis, a chronic inflammatory and lipid-driven condition affecting the vascular endothelium.1. Endothelial DamageThe process begins with damage to the vascular endothelium, which serves as a protective barrier between the blood and the vessel...
23
Acute Coronary Syndrome II: Pathophysiology and Clinical Manifestations01:19

Acute Coronary Syndrome II: Pathophysiology and Clinical Manifestations

35
The pathophysiology of Acute Coronary Syndrome [ACD] involves several key processes:The main underlying cause of ACD is atherosclerosis, a chronic inflammatory disease characterized by the buildup of lipid-laden plaques within the coronary arteries.As the atherosclerotic plaque grows in the coronary artery, it may become unstable due to the formation of a lipid-rich core and a thin fibrous cap. Inflammatory cells within the plaque, such as macrophages, secrete enzymes that degrade the...
35
Anticoagulant Drugs: Low-Molecular-Weight Heparins01:30

Anticoagulant Drugs: Low-Molecular-Weight Heparins

834
Hemostasis is a crucial process that prevents excessive blood loss from damaged blood vessels. It involves various mechanisms such as vasoconstriction, platelet adhesion and activation, and fibrin formation. The importance of each mechanism depends on the type of vessel injury. In contrast, thrombosis is the abnormal formation of a blood clot within the blood vessels, leading to potential complications if the clot obstructs blood flow. Thrombosis can be caused by increased coagulability of the...
834
Atherosclerosis I: Introduction01:30

Atherosclerosis I: Introduction

35
Atherosclerosis is a progressive disorder characterized by the buildup of plaques on the arterial inner wall, causing them to narrow and harden over time. These plaques comprise lipids, calcium, blood components, carbohydrates, and fibrous tissue. The process primarily affects the intima of large and medium-sized arteries, reducing blood flow in any artery.Etiology and risk factorsThe cause of atherosclerosis is multifactorial, involving a complex interplay among endothelial injury, lipid...
35

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Gut Microbiome-Modulating therapeutics and lipid profile in metabolic syndrome: A systematic review and meta-analysis of clinical trials.

Clinical nutrition ESPEN·2026
Same author

Significance of Gastrointestinal Histiocytosis: A Case Series and Literature Review.

Cureus·2026
Same author

Safety and performance of small bowel capsule endoscopy in patients with surgically altered gastrointestinal anatomy.

Scandinavian journal of gastroenterology·2026
Same author

Endoscopic Retrieval of a Migrated Plastic Pigtail Stent in a Patient With Gastric Bypass Surgery Using Novel Endoscopic Cutting Device.

ACG case reports journal·2026
Same author

Circulating Inflammation-Related Proteins Linked to Corneal Neuroimmune Measures, Neuropathy Deficits, and Symptoms in Type 2 Diabetes.

Journal of the peripheral nervous system : JPNS·2026
Same author

An Endoscopic Approach to a Pouch Stricture After Failed Needle Knife Stricturotomy.

ACG case reports journal·2026

Related Experiment Video

Updated: Aug 20, 2025

Catheter Ablation in Combination With Left Atrial Appendage Closure for Atrial Fibrillation
28:13

Catheter Ablation in Combination With Left Atrial Appendage Closure for Atrial Fibrillation

Published on: February 26, 2013

33.5K

Alcohol and Atrial Fibrillation: A Pathophysiologic Perspective.

Risheek Kaul1, Ridhima Kaul2, Pradipta Paul2

  • 1From the Department of Cardiology, Westchester Medical Center/New York Medical College, Valhalla, NY.

Cardiology in Review
|November 18, 2022
PubMed
Summary

Alcohol consumption increases the risk of new-onset atrial fibrillation (AF), a common heart rhythm disorder. This review explores the scientific evidence linking alcohol intake to AF and its underlying biological mechanisms.

More Related Videos

High-Resolution Endocardial and Epicardial Optical Mapping in a Sheep Model of Stretch-Induced Atrial Fibrillation
09:17

High-Resolution Endocardial and Epicardial Optical Mapping in a Sheep Model of Stretch-Induced Atrial Fibrillation

Published on: July 29, 2011

14.9K
Optimization of Transesophageal Atrial Pacing to Assess Atrial Fibrillation Susceptibility in Mice
08:05

Optimization of Transesophageal Atrial Pacing to Assess Atrial Fibrillation Susceptibility in Mice

Published on: June 29, 2022

2.9K

Related Experiment Videos

Last Updated: Aug 20, 2025

Catheter Ablation in Combination With Left Atrial Appendage Closure for Atrial Fibrillation
28:13

Catheter Ablation in Combination With Left Atrial Appendage Closure for Atrial Fibrillation

Published on: February 26, 2013

33.5K
High-Resolution Endocardial and Epicardial Optical Mapping in a Sheep Model of Stretch-Induced Atrial Fibrillation
09:17

High-Resolution Endocardial and Epicardial Optical Mapping in a Sheep Model of Stretch-Induced Atrial Fibrillation

Published on: July 29, 2011

14.9K
Optimization of Transesophageal Atrial Pacing to Assess Atrial Fibrillation Susceptibility in Mice
08:05

Optimization of Transesophageal Atrial Pacing to Assess Atrial Fibrillation Susceptibility in Mice

Published on: June 29, 2022

2.9K

Area of Science:

  • Cardiology
  • Electrophysiology
  • Public Health

Background:

  • Atrial fibrillation (AF) is a prevalent cardiac arrhythmia with substantial morbidity and mortality.
  • The global burden of AF is projected to rise, impacting patients and healthcare systems.
  • Existing research indicates a dose-response correlation between alcohol consumption and new-onset AF.

Approach:

  • This review synthesizes current scientific literature on the association between alcohol consumption and AF.
  • It examines the pathophysiological mechanisms underlying this relationship.
  • The review aims to elucidate how alcohol affects cardiac function and contributes to AF.

Key Points:

  • Alcohol intake demonstrates a dose-dependent relationship with the incidence of new-onset AF.
  • Mechanisms include alterations in atrial conduction, direct cellular effects on myocytes, and autonomic nervous system dysregulation.
  • This link is observed independently of common risk factors like sex and age.

Conclusions:

  • Alcohol consumption is a significant modifiable risk factor for atrial fibrillation.
  • Understanding the pathophysiological pathways is crucial for developing targeted prevention and management strategies.
  • Further research into the specific cellular and neural effects of alcohol on the atria is warranted.