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A novel thyroid hormone receptor encoded by a cDNA clone from a human testis library

D Benbrook1, M Pfahl

  • 1Cancer Research Center, La Jolla Cancer Research Foundation, CA 92037.

Science (New York, N.Y.)
|November 6, 1987
PubMed

Insights

Researchers identified a novel human thyroid hormone receptor, erbA-T, by screening a testis cDNA library. This discovery suggests multiple human thyroid hormone receptors with distinct tissue and gene-activating specificities exist.

Area of Science:

  • Molecular Biology
  • Endocrinology
  • Genetics

Background:

  • The c-erbA gene family encodes transcriptional regulators, including steroid hormone and vitamin D3 receptors.
  • The v-erbA oncogene product is also part of this multigene family.
  • Thyroid hormone receptors are crucial for regulating gene expression.

Purpose of the Study:

  • To identify novel human genes related to the c-erbA family.
  • To characterize a newly isolated human gene product for its potential role as a thyroid hormone receptor.
  • To investigate the existence of multiple thyroid hormone receptors in humans.

Main Methods:

  • Screening of a human complementary DNA (cDNA) testis library using a v-erbA DNA probe.
  • Isolation and sequencing of a specific clone, erbA-T-1.
  • Analysis of the encoded protein's (erbA-T) homology with known c-erbA proteins.
  • Binding assays to determine the interaction of erbA-T with 3,5,3'-triiodo-L-thyronine (T3).

Main Results:

  • A novel human cDNA clone, erbA-T-1, encoding a 490-amino acid protein (erbA-T) was isolated.
  • The erbA-T protein exhibits high homology to chicken c-erbA and human c-erbA-beta proteins.
  • erbA-T specifically binds 3,5,3'-triiodo-L-thyronine (T3) with a high affinity (Kd = 3.8 +/- 0.2 x 10(-10) M).

Conclusions:

  • The data indicate the existence of at least one additional thyroid hormone receptor in humans, designated erbA-T.
  • This novel receptor may possess distinct tissue-specific and gene-activating functions compared to other known thyroid hormone receptors.
  • The findings suggest a more complex regulatory network for thyroid hormone action in humans.

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