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Listeriosis in diacetoxyscirpenol-treated mice

R L Ziprin1, D E Corrier

  • 1Veterinary Toxicology and Entomology Research Laboratory, USDA, College Station, TX 77841.

Insights

Diacetoxyscirpenol (DAS), a mycotoxin, increased mortality and Listeria monocytogenes bacterial load in mice. DAS also caused thymus atrophy and lymphocyte depletion, impacting immune response to infection.

Area of Science:

  • Immunotoxicology
  • Microbiology
  • Mycotoxicology

Background:

  • Trichothecene mycotoxins, such as diacetoxyscirpenol (DAS), are contaminants found in food and feed.
  • These mycotoxins can pose a risk to animal and human health by affecting the immune system.

Purpose of the Study:

  • To investigate the immunotoxic effects of diacetoxyscirpenol (DAS) on the course of Listeria monocytogenes infection in mice.
  • To determine the impact of DAS exposure on host susceptibility, mortality, and immune cell populations.

Main Methods:

  • Mice were administered diacetoxyscirpenol (DAS) via oral gavage at specific time points before intraperitoneal inoculation with Listeria monocytogenes.
  • Mortality rates and splenic bacterial loads were monitored to assess infection severity.
  • Thymus weights and lymphocyte distribution in thymus and spleen were histologically examined.

Main Results:

  • Mice treated with 3 mg/kg DAS two days before Listeria challenge exhibited increased mortality and higher splenic bacterial counts.
  • DAS treatment led to reduced thymus weights and significant lymphocyte depletion in the thymus cortex and splenic lymphoid tissues.
  • A single dose of 4 mg/kg DAS given six days prior to infection did not alter mortality.
  • DAS-treated mice showed significantly increased neutrophil populations compared to controls.

Conclusions:

  • Pre-infection exposure to diacetoxyscirpenol (DAS) at a dose of 3 mg/kg impairs the host immune response, leading to increased susceptibility to Listeria monocytogenes infection.
  • DAS-induced immunosuppression is characterized by thymus atrophy, lymphocyte depletion, and altered neutrophil response.
  • The timing and dosage of DAS exposure are critical factors influencing its immunotoxic effects.

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