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Published on: December 14, 2020
Architecture and matrix assembly determinants of Bordetella pertussis biofilms on primary human airway epithelium
Audra R Fullen1, Jessica L Gutierrez-Ferman1, Rachael E Rayner2
1The Department of Microbial Infection and Immunity, The Ohio State University Wexner Medical Center, Columbus, Ohio, United States of America.
Abstract:
Traditionally, whooping cough or pertussis caused by the obligate human pathogen Bordetella pertussis (Bp) is described as an acute disease with severe symptoms. However, many individuals who contract pertussis are either asymptomatic or show very mild symptoms and yet can serve as carriers and sources of bacterial transmission. Biofilms are an important survival mechanism for bacteria in human infections and disease. However, bacterial determinants that drive biofilm formation in humans are ill-defined. In the current study, we show that Bp infection of well-differentiated primary human bronchial epithelial cells leads to formation of bacterial aggregates, clusters, and highly structured biofilms which are colocalized with cilia. These findings mimic observations from pathological analyses of tissues from pertussis patients. Distinct arrangements (mono-, bi-, and tri-partite) of the polysaccharide Bps, extracellular DNA, and bacterial cells were visualized, suggesting complex heterogeneity in bacteria-matrix interactions. Analyses of mutant biofilms revealed positive roles in matrix production, cell cluster formation, and biofilm maturity for three critical Bp virulence factors: Bps, filamentous hemagglutinin, and adenylate cyclase toxin. Adherence assays identified Bps as a new Bp adhesin for primary human airway cells. Taken together, our results demonstrate the multi-factorial nature of the biofilm extracellular matrix and biofilm development process under conditions mimicking the human respiratory tract and highlight the importance of model systems resembling the natural host environment to investigate pathogenesis and potential therapeutic strategies.
Insights
Bordetella pertussis forms structured biofilms in human airways, mimicking patient infections. Key virulence factors like Bps are crucial for biofilm development and bacterial adherence, offering insights into pertussis pathogenesis.
Area of Science:
- Microbiology
- Pathogenesis
- Bacterial biofilms
Background:
- Pertussis (whooping cough) is caused by Bordetella pertussis (Bp).
- Asymptomatic or mild infections can lead to transmission.
- Bacterial biofilm formation determinants in human infections are poorly understood.
Purpose of the Study:
- To investigate Bordetella pertussis biofilm formation in a human airway model.
- To identify bacterial factors contributing to biofilm development and structure.
- To understand the role of Bp virulence factors in biofilm formation and adherence.
Main Methods:
- Infection of differentiated primary human bronchial epithelial cells with Bp.
- Visualization of bacterial aggregates, biofilms, and matrix components (Bps, eDNA).
- Analysis of Bp mutant strains to assess the role of virulence factors in biofilm formation.
- Adherence assays to evaluate Bps as an adhesin.
Main Results:
- Bp forms structured biofilms colocalized with cilia, resembling pathological findings.
- Heterogeneous bacterial-matrix interactions involving polysaccharide Bps, eDNA, and bacterial cells were observed.
- Bps, filamentous hemagglutinin, and adenylate cyclase toxin positively regulate biofilm matrix production, cell clustering, and maturity.
- Bps was identified as a novel adhesin for human airway cells.
Conclusions:
- Biofilm development in Bp is a multi-factorial process involving complex extracellular matrix interactions.
- Virulence factors play critical roles in Bp biofilm structure and maturation.
- Human airway cell models are essential for studying Bp pathogenesis and developing therapeutic strategies.
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