Related Experiment Video
Updated: Aug 8, 2025

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats
Published on: November 20, 2015
Objectives:
The aim of the presented work is to summarize the current knowledge about the pathophysiology of preterm birth in connection with premature amniotic fluid.
Methods:
To analyze current knowledge and our own experiences regarding of preterm prelabour rupture of membranes.
Conclusion:
The most important factor influencing neonatal morbidity and mortality is gestational age. Early neonatal sepsis occurs with high risk after premature amniotic fluid outflow, associated with inflammatory complications.
More Related Videos
08:50A Murine Model of Fetal Exposure to Maternal Inflammation to Study the Effects of Acute Chorioamnionitis on Newborn Intestinal Development
Published on: June 24, 2020
05:31Author Spotlight: Modeling an Aspect of Preeclampsia in Female Mice Using Hypoxic Human Placenta-Derived Small Extracellular Vesicles
Published on: January 26, 2024
Related Concept Videos
Mitral Valve Prolapse III: Nursing Management
Pulmonary Embolism I: Introduction
Teratogenicity
Types of Membrane Protrusions
The microvilli, an example of stable protrusions, are finger-like projections...
Esophageal Perforation-I: Introduction
The location of esophageal perforation can vary, occurring anywhere along the esophagus....
Olefin Metathesis Polymerization: Ring-Opening Metathesis Polymerization (ROMP)