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Thrombosis at mountain altitudes.

S A Cucinell1, C M Pitts

  • 1Department of Clinical Investigation, Tripler Army Medical Center, HI 96859-6709.

Aviation, Space, and Environmental Medicine
|November 1, 1987
PubMed
Summary
This summary is machine-generated.

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Mountain travel may increase blood clot risk, potentially leading to high-altitude pulmonary edema. However, current lab tests are insufficient to confirm this hypercoagulability mechanism in mountain sickness.

Area of Science:

  • Physiology
  • Altitude Medicine
  • Hematology

Background:

  • High-altitude pulmonary edema (HAPE) is often associated with pulmonary vessel clots.
  • Thrombophlebitis and cerebral emboli appear frequent at high altitudes.
  • Mountain travel may be linked to a hypercoagulable state.

Purpose of the Study:

  • To critically analyze data suggesting mountain travel predisposes individuals to hypercoagulability.
  • To investigate the role of thrombosis in mountain sickness.

Main Methods:

  • Critical analysis of existing data and literature.
  • Review of laboratory techniques for characterizing hypercoagulability.

Main Results:

  • Thrombosis might be a late complication of certain mountain sickness forms.

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  • Current laboratory methods are inadequate for defining hypercoagulability mechanisms at altitude.

    • Conclusions:

    • While mountain travel may be associated with increased clotting risk, definitive mechanisms remain unclear.
    • Further research is needed to develop reliable methods for assessing altitude-induced hypercoagulability.