[Sudden cardiac death after cardioversion]
Felix Bertram1, Jochen Buchholz2
1Klinik für Innere Medizin, Evangelisches Krankenhaus Oberhausen (Rheinland), Oberhausen, Deutschland, Virchowstr. 20, 46047 Oberhausen.
Insights
A patient with heart failure experienced a fatal outcome due to QT interval prolongation and torsade de pointes after cardioversion. Impaired left ventricular function and atrial ectopy were identified as the primary causes.
Area of Science:
- Cardiology
- Electrophysiology
Background:
- Atrial tachycardia with reduced left ventricular ejection fraction (LVEF) is a significant cause of heart failure.
- Cardioversion is a common procedure to restore normal heart rhythm.
Observation:
- An 83-year-old female patient with heart failure presented with atrial tachycardia and reduced LVEF.
- The patient underwent cardioversion for rhythm restoration.
Findings:
- Post-cardioversion Holter monitoring revealed massive QT interval prolongation.
- This prolongation led to torsade de pointes, a life-threatening ventricular arrhythmia.
- Impaired left ventricular function and atrial ectopy were identified as the sole contributors to QT prolongation.
Implications:
- This case highlights the critical risk of QT prolongation and torsade de pointes in patients with compromised left ventricular function.
- Careful electrophysiological assessment and monitoring are crucial before and after cardioversion in such high-risk patients.
- Understanding the interplay between cardiac function, atrial ectopy, and proarrhythmic events is vital for preventing adverse outcomes.
Abstract:
An 83-year-old woman with heart failure due to atrial tachycardia with reduced left ventricular ejection fraction died after cardioversion. Holter monitoring showed a massive prolongation of the QT interval resulting in torsade de pointe tachycardia with lethal outcome. The only reason of the QT prolongation was impaired left ventricular (LV) function and atrial ectopy.
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