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Thyroidectomy of SHR: effects on ventricular relaxation and on SR calcium uptake activity

Insights

Hypothyroidism impairs cardiac relaxation and calcium uptake in spontaneously hypertensive rats (SHR) and Wistar-Kyoto rats (WKY). Combined hypothyroidism and SHR exacerbates these cardiac dysfunctions, highlighting a significant correlation between mechanical relaxation and SR calcium uptake.

Area of Science:

  • Cardiovascular Physiology
  • Endocrinology
  • Molecular Cardiology

Background:

  • Ventricular hypertrophy and hypothyroidism both impair cardiac muscle relaxation and sarcoplasmic reticulum (SR) calcium uptake.
  • Previous studies indicated hypothyroidism does not reverse ventricular hypertrophy in spontaneously hypertensive rats (SHR).

Purpose of the Study:

  • To investigate the effects of induced hypothyroidism on cardiac relaxation and SR calcium uptake in SHR and Wistar-Kyoto rat (WKY) controls.
  • To determine if hypothyroidism exacerbates cardiac dysfunction in SHR.

Main Methods:

  • Thyroidectomy was performed for 8 weeks on SHR and WKY rats.
  • Cardiac relaxation was assessed in ejecting hearts by measuring left ventricular pressure changes.
  • SR calcium uptake activity was measured in vitro.

Main Results:

  • Euthyroid SHR exhibited impaired relaxation and reduced SR calcium uptake compared to euthyroid WKY rats.
  • Thyroidectomy similarly reduced relaxation and SR calcium uptake in both SHR and WKY strains.
  • The most depressed cardiac function was observed in the hypothyroid SHR group.

Conclusions:

  • Hypothyroidism significantly impairs cardiac relaxation and SR calcium uptake in both normotensive and hypertensive rat models.
  • The combination of SHR and hypothyroidism leads to the most severe cardiac dysfunction.
  • A strong correlation exists between ex vivo mechanical relaxation and in vitro SR calcium uptake rates.

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