Post myocardial infarction ventricular septal rupture-protracted hemolysis due to missed additional ventricular

Vivek Bagaria1, Channabasavaraj Shivalingaiah Hiremath1

  • 1Department of Cardiothoracic and Vascular Surgery, Sri Sathya Sai Institute of Higher Medical Sciences, Whitefield, Bangalore, 560066 India.

Insights

Protracted hemolysis after ventricular septal rupture (VSR) device closure can necessitate surgical intervention. A case revealed a muscular band dividing the VSR, causing a residual shunt and persistent hemolysis.

Area of Science:

  • Cardiology
  • Interventional Cardiology
  • Cardiac Surgery

Background:

  • Transcatheter device closure is a common treatment for post-myocardial infarction ventricular septal rupture (VSR).
  • Hemolysis is a recognized complication, typically resolving spontaneously.
  • Persistent hemolysis may indicate residual shunting or device-related issues.

Observation:

  • A patient experienced prolonged hemolysis following VSR device closure.
  • Surgical intervention was required for device retrieval and VSR closure.
  • A muscular band was identified, dividing the VSR into two distinct defects.

Findings:

  • The muscular band created a residual shunt through the second VSR defect.
  • This residual shunt was the likely cause of the protracted hemolysis.
  • Surgical patch closure successfully addressed the residual defect and resolved hemolysis.

Implications:

  • This case highlights the importance of thorough anatomical assessment before VSR device closure.
  • Unrecognized complex VSR anatomy, such as septation, can lead to device failure and complications.
  • Surgical revision may be necessary in cases of persistent hemolysis due to residual shunting.
  • This emphasizes the need for careful follow-up after transcatheter VSR closure.

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