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Published on: September 9, 2020
[Left Ventricular Plasty for Left Ventricular Aneurysm Following Takotsubo Cardiomyopathy]
Masahide Shichijo1, Yuji Katayama, Hiroyuki Morokuma
1Department of Cardiovascular Surgery, Fukuoka Tokushukai Hospital, Kasuga, Japan.
Insights
Takotsubo cardiomyopathy, a form of heart failure, can lead to severe left ventricular dysfunction. This case highlights successful left ventricular plasty for managing advanced takotsubo cardiomyopathy with apical aneurysm.
Area of Science:
- Cardiology
- Cardiovascular Surgery
Context:
- Takotsubo cardiomyopathy (TTC), or stress-induced cardiomyopathy, is characterized by transient left ventricular dysfunction.
- This condition can mimic myocardial infarction but lacks obstructive coronary artery disease.
Purpose:
- To describe a case of advanced takotsubo cardiomyopathy with left ventricular apical aneurysm and intractable heart failure.
- To report the successful surgical management using left ventricular plasty.
Summary:
- An 82-year-old woman presented with chest pain, diagnosed with takotsubo cardiomyopathy.
- Despite medical management, she developed intractable heart failure and an apical aneurysm with pericardial effusion.
- Left ventricular plasty was performed, resulting in significant reduction of ventricular volume and a favorable outcome.
Impact:
- This case demonstrates the potential efficacy of left ventricular plasty in select patients with severe, refractory takotsubo cardiomyopathy and apical aneurysm.
- Surgical intervention may be a viable option for managing complications of takotsubo cardiomyopathy unresponsive to medical therapy.
Abstract:
An 82-years-old woman was admitted with sudden chest pain. Coronary angiography did not reveal any significant stenosis, but left ventriculography showed akinesis and ballooning of the apex with a hyperkinetic basal segment, suggestive of takotsubo cardiomyopathy. She suffered intractable heart failure, and laboratory data, electrocardiogram signs of ST-segment elevation and left ventricular( LV) apical dysfunction failed to show improvement. LV plasty was performed on 106th day after the onset because of aneurysmal change of the left ventricular apex myocardium, unremitting sanguineous pericardial effusion, and anemia. She had a good postoperative course and end-diastolic ventricular volume index was remarkably reduced from 144 to 55 ml/m2. She discharged home on the 38th postoperative day.
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