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Depot triamcinolone-induced glaucoma.

D W Mills, L F Siebert, D B Climenhaga

Canadian Journal of Ophthalmology. Journal Canadien D'Ophtalmologie

|June 1, 1986

Summary

This summary is machine-generated.

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Recalcitrant ocular hypertension after triamcinolone acetonide injections resolved with surgical removal of residual steroid plaques. This intervention effectively treated persistent glaucoma in two reported cases.

Area of Science:

Ophthalmology
Pharmacology

Background:

Subconjunctival injections of triamcinolone acetonide are used to treat various ocular inflammatory conditions.
Persistent ocular hypertension can be a side effect of corticosteroid therapy.

Observation:

Two patients experienced prolonged ocular hypertension, lasting 6.5 and 10 months, after receiving subconjunctival triamcinolone acetonide injections.
A visible whitish plaque of residual steroid was noted in the subconjunctival space in both cases.

Findings:

Surgical excision of the subconjunctival steroid plaque led to prompt resolution of ocular hypertension in both patients.
This suggests a direct link between the residual steroid depot and sustained elevated intraocular pressure.

Implications:

Related Experiment Videos

Surgical removal of steroid plaques may be an effective treatment for refractory ocular hypertension post-injection.
This highlights the importance of monitoring for and managing steroid-induced ocular hypertension, potentially through surgical intervention.