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Related Concept Videos

Acute Coronary Syndrome III: Diagnostic Studies01:30

Acute Coronary Syndrome III: Diagnostic Studies

9
Diagnosing acute coronary syndrome or ACS begins with a thorough patient history. Notable symptoms include central, crushing chest pain radiating to the left arm, neck, jaw, or back, along with shortness of breath, sweating (diaphoresis), nausea, vomiting, dizziness, and palpitations.It is crucial to note any history of cardiac illnesses and assess risk factors, including age, gender, smoking, hypertension, diabetes, hyperlipidemia, and a sedentary lifestyle.During physical examination, vital...
9
Coronary Artery Disease IV: Preventive Measures01:26

Coronary Artery Disease IV: Preventive Measures

23
Effective preventive measures for coronary artery disease (CAD) focus on controlling modifiable risk factors, including cholesterol abnormalities and lifestyle changes.Cholesterol ManagementFirst, the Mediterranean diet and the American Heart Association advocate for maintaining low-density lipoprotein (LDL) cholesterol levels below 100 mg/dL, with a more stringent recommendation of below 70 mg/dL for individuals at high risk. LDL cholesterol, often termed "bad cholesterol," can lead to the...
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Acute Coronary Syndrome IV: Interprofessional Care01:28

Acute Coronary Syndrome IV: Interprofessional Care

11
IntroductionThe management of Acute Coronary Syndrome (ACS) aims to minimize myocardial damage, preserve myocardial function, and prevent complications.Initial ManagementInpatient management involves continuous cardiac monitoring, preferably in an ICU, focusing on blood pressure, serum sodium, potassium, and creatinine levels, and urine output. Ongoing pharmacologic management is crucial for stabilizing the patient.Supplemental Oxygen: Administer supplemental oxygen if oxygen saturation is...
11
Coronary Artery Disease V: Interprofessional Care01:27

Coronary Artery Disease V: Interprofessional Care

9
Interprofessional care for coronary artery disease includes pharmacological therapy and revascularization procedures.Pharmacological therapy for Coronary Artery Disease (CAD) aims to manage symptoms, prevent complications, and improve patient outcomes through various classes of medications:Antiplatelet Agents:Aspirin and Clopidogrel: These medications inhibit platelet aggregation, preventing blood clots, which is crucial for avoiding heart attacks and strokes. Doctors often prescribe these...
9
Acute Coronary Syndrome II: Pathophysiology and Clinical Manifestations01:19

Acute Coronary Syndrome II: Pathophysiology and Clinical Manifestations

20
The pathophysiology of Acute Coronary Syndrome [ACD] involves several key processes:The main underlying cause of ACD is atherosclerosis, a chronic inflammatory disease characterized by the buildup of lipid-laden plaques within the coronary arteries.As the atherosclerotic plaque grows in the coronary artery, it may become unstable due to the formation of a lipid-rich core and a thin fibrous cap. Inflammatory cells within the plaque, such as macrophages, secrete enzymes that degrade the...
20
Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care01:29

Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care

10
Diagnosing Pulmonary EmbolismDiagnosing pulmonary embolism (PE) involves clinical assessment and advanced imaging tests. The preferred diagnostic tool is the spiral (helical) CT scan or CT angiography (CTA), which uses intravenous contrast media to visualize the pulmonary vasculature and identify emboli.A ventilation-perfusion (V/Q) scan is an alternative for patients unable to receive contrast media. This scan includes both perfusion and ventilation scanning. Perfusion scanning involves...
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  1. Home
  3. Research Domains
  5. Biomedical And Clinical Sciences
  7. Oncology And Carcinogenesis
  9. Predictive And Prognostic Markers
  11. Smoking And Cardiovascular Outcomes After Percutaneous Coronary Intervention: A Korean Study.
  1. Home
  3. Research Domains
  5. Biomedical And Clinical Sciences
  7. Oncology And Carcinogenesis
  9. Predictive And Prognostic Markers
  11. Smoking And Cardiovascular Outcomes After Percutaneous Coronary Intervention: A Korean Study.

Related Experiment Video

Left Anterior Descending Coronary Artery Ligation for Ischemia-Reperfusion Research: Model Improvement via Technical Modifications and Quality Control
05:41

Left Anterior Descending Coronary Artery Ligation for Ischemia-Reperfusion Research: Model Improvement via Technical Modifications and Quality Control

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Smoking and cardiovascular outcomes after percutaneous coronary intervention: a Korean study.

You-Jeong Ki1, Kyungdo Han2, Hyo-Soo Kim3,4

  • 1Cardiovascular Center, Uijeongbu Eulji Medical Center, Uijeongbu-si, Gyeonggi-do, Republic of Korea.

European Heart Journal
|September 27, 2023

View abstract on PubMed

Summary
This summary is machine-generated.

Smoking after percutaneous coronary intervention (PCI) significantly increases adverse cardiovascular events. However, quitting before 20 pack-years (PYs) of exposure post-PCI offers outcomes comparable to non-smokers.

Keywords:
Cardiovascular eventCessationPercutaneous coronary interventionSmoking

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Coronary Progenitor Cells and Soluble Biomarkers in Cardiovascular Prognosis after Coronary Angioplasty
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Aggravation of Myocardial Ischemia upon Particulate Matter Exposure in Atherosclerosis Animal Model
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Area of Science:

  • Cardiology
  • Public Health
  • Smoking Cessation Research

Background:

  • Smoking is a major risk factor for cardiovascular disease.
  • Percutaneous coronary intervention (PCI) is a common procedure for managing coronary artery disease.
  • The impact of smoking status and cessation on outcomes after PCI requires further investigation.

Purpose of the Study:

  • To investigate the association between smoking status and cardiovascular outcomes after PCI.
  • To evaluate the effect of smoking cessation on major adverse cardiovascular and cerebrovascular events (MACCE) in patients who underwent PCI.
  • To determine if the amount of smoking exposure influences the benefits of cessation.

Main Methods:

  • A nationwide cohort of 74,471 patients undergoing PCI (2009-2016) was analyzed using Korean National Health Insurance System data.
Tobacco
  • Patients were categorized into non-smokers, ex-smokers, and current smokers based on post-PCI health check-ups.
  • Major adverse cardiovascular and cerebrovascular event (MACCE) rates were compared, with a focus on smoking cessation effects in a subset of 31,887 patients with pre- and post-PCI data.

    • Main Results:

    • Current smokers exhibited a 19.8% higher risk of MACCE compared to non-smokers (aHR 1.198).
    • Ex-smokers showed a MACCE rate comparable to non-smokers (aHR 1.036).
    • Among patients who quit smoking post-PCI, those with less than 20 pack-years (PYs) of cumulative exposure had outcomes similar to persistent non-smokers. However, quitters with ≥20 PYs had risks comparable to persistent smokers.

    Conclusions:

    • Smoking is linked to increased adverse outcomes in patients undergoing PCI.
    • Smoking cessation after PCI, particularly with less than 20 pack-years of exposure, is associated with cardiovascular risk comparable to that of non-smokers.
    • Early smoking cessation is crucial for improving cardiovascular outcomes post-PCI.