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Pramisha Kharel1, Chen Jia1, Kamal Raj Dhital2
1From the Department of Rheumatology and Immunology, Shanghai Skin Disease Hospital, Tongji University School of Medicine, Shanghai, China.
Tumour necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) and its receptor fibroblast growth factor-inducible 14 (Fn14) pathway are implicated in skin inflammation and autoimmune diseases. This review explores TWEAK/Fn14
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