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Increased insulin responsiveness in endotoxicosis.

J P Filkins, D P Figlewicz

Circulatory Shock

|January 1, 1979

Summary

Endotoxemia significantly alters glucose metabolism and insulin sensitivity in rats. Tissues show increased glucose oxidation, impacting overall glucose homeostasis during endotoxicosis.

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Increased insulin responsiveness in endotoxicosis.

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Loss of islet sympathetic nerves and impairment of glucagon secretion in the NOD mouse: relationship to invasive insulitis.

Expression of receptors for insulin and leptin in the ventral tegmental area/substantia nigra (VTA/SN) of the rat.

Intraventricular insulin decreases kappa opioid-mediated sucrose intake in rats.

Effects of repetitive hypoglycemia on neuroendocrine response and brain tyrosine hydroxylase activity in the rat.

Related Experiment Videos

Increased insulin responsiveness in endotoxicosis.

Related Concept Videos

Related Articles

Effect of recurrent yohimbine on immediate and post-hoc behaviors, stress hormones, and energy homeostatic parameters.

Effect of moderate intake of sweeteners on metabolic health in the rat.

Loss of islet sympathetic nerves and impairment of glucagon secretion in the NOD mouse: relationship to invasive insulitis.

Expression of receptors for insulin and leptin in the ventral tegmental area/substantia nigra (VTA/SN) of the rat.

Intraventricular insulin decreases kappa opioid-mediated sucrose intake in rats.

Effects of repetitive hypoglycemia on neuroendocrine response and brain tyrosine hydroxylase activity in the rat.

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