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Updated: Jul 12, 2025

Real-Time Monitoring of Aurora kinase A Activation using Conformational FRET Biosensors in Live Cells
Published on: July 30, 2020
Computational completion of the Aurora interaction region of N-Myc in the Aurora a kinase complex
Pinar Altiner1, Süleyman Selim Çınaroğlu2, Ahmet Can Timucin3
1Institut de Pharmacologie et de Biologie Structurale (IPBS), CNRS, Université Toulouse III - Paul Sabatier (UT3), 31077, Toulouse, France.
Abstract:
Inhibiting protein-protein interactions of the Myc family is a viable pharmacological strategy for modulation of the levels of Myc oncoproteins in cancer. Aurora A kinase (AurA) and N-Myc interaction is one of the most attractive targets of this strategy because formation of this complex blocks proteasomal degradation of N-Myc in neuroblastoma. Two crystallization studies have captured this complex (PDB IDs: 5g1x, 7ztl), partially resolving the AurA interaction region (AIR) of N-Myc. Prompted by the missing N-Myc fragment in these crystal structures, we modeled the complete structure between AurA and N-Myc, and comprehensively analyzed how the incomplete and complete N-Myc behave in complex by molecular dynamics simulations. Molecular dynamics simulations of the incomplete PDB complex (5g1x) repeatedly showed partial dissociation of the short N-Myc fragment (61-89) from the kinase. The missing N-Myc (19-60) fragment was modeled utilizing the N-terminal lobe of AurA as the protein-protein interaction surface, wherein TPX2, a well-known partner of AurA, also binds. Binding free energy calculations along with flexibility analysis confirmed that the complete AIR of N-Myc stabilizes the complex, accentuating the N-terminal lobe of AurA as a binding site for the missing N-Myc fragment (19-60). We further generated additional models consisting of only the missing N-Myc (19-60), and the fused form of TPX2 (7-43) and N-Myc (61-89). These partners also formed more stable interactions with the N-terminal lobe of AurA than did the incomplete N-Myc fragment (61-89) in the 5g1x complex. Altogether, this study provides structural insights into the involvement of the N-terminus of the AIR of N-Myc and the N-terminal lobe of AurA in formation of a stable complex, reflecting its potential for effective targeting of N-Myc.
Insights
Targeting the Myc oncoprotein in cancer is crucial. This study reveals how the complete N-Myc protein structure stabilizes its complex with Aurora A kinase, offering new therapeutic strategies.
Area of Science:
- Biochemistry
- Structural Biology
- Cancer Biology
Background:
- Inhibiting Myc oncoprotein interactions is a key cancer therapy strategy.
- Aurora A kinase (AurA) and N-Myc interaction blocks N-Myc degradation in neuroblastoma.
- Existing crystal structures (5g1x, 7ztl) show incomplete N-Myc interaction regions (AIR).
Purpose of the Study:
- To model and analyze the complete AurA-N-Myc complex structure.
- To investigate the role of the N-terminal AIR of N-Myc in complex stability.
- To identify potential therapeutic targets for neuroblastoma.
Main Methods:
- Protein structure modeling of the complete AurA-N-Myc complex.
- Molecular dynamics simulations to analyze complex behavior.
- Binding free energy calculations and flexibility analysis.
Main Results:
- The incomplete N-Myc fragment (61-89) showed partial dissociation in simulations.
- The N-terminal lobe of AurA was identified as a binding site for the modeled N-Myc fragment (19-60).
- The complete AIR of N-Myc significantly stabilizes the AurA complex.
Conclusions:
- The N-terminus of N-Myc's AIR and AurA's N-terminal lobe are critical for stable complex formation.
- This interaction offers a potential target for modulating N-Myc levels in cancer.
- The findings provide structural insights for developing novel anti-cancer therapeutics.
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