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Related Experiment Videos

REM latency and core temperature relationships in primary depression.

D H Avery, G Wildschiødtz, R G Smallwood

    Acta Psychiatrica Scandinavica
    |September 1, 1986
    PubMed
    Summary

    Depressed patients exhibit shorter REM latency and higher body temperatures. These measures normalized with recovery, suggesting a link between temperature regulation and sleep disturbances in depression.

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    Nocturnal sweating and temperature in depression.

    Acta psychiatrica Scandinavica·1999

    Area of Science:

    • Neuroscience
    • Sleep Medicine
    • Psychiatry

    Background:

    • Depression is associated with sleep disturbances, including altered REM sleep.
    • Body temperature regulation may be disrupted in individuals with depression.

    Purpose of the Study:

    • To investigate the relationship between REM latency and body temperature (rectal and ear canal) in depressed patients compared to controls.
    • To examine how these parameters change with recovery from depression.

    Main Methods:

    • Simultaneous measurement of REM latency, rectal temperature, and ear canal temperature.
    • Study included 11 healthy controls and 9 depressed patients (7 studied during recovery).

    Main Results:

    • Depressed patients showed shorter REM latency and higher nocturnal rectal and ear canal temperatures compared to controls.

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  • These physiological markers normalized with recovery from depression.
  • A negative correlation was found between REM latency and nocturnal rectal temperature in both depressed patients and all subjects.
  • A negative correlation was also observed between REM latency and nocturnal ear canal temperature in the control group.

    • Conclusions:

    • Altered body temperature regulation and REM sleep patterns are characteristic of depression.
    • These physiological abnormalities appear to be reversible with successful treatment and recovery.
    • The findings suggest a potential link between thermoregulation and sleep disturbances in the pathophysiology of depression.