KIAA1429 facilitates metastasis via m6A-YTHDC1-dependent RND3 down-regulation in hepatocellular carcinoma cells
Meihua Shan1, Dong Liu1, Liangbo Sun1
1Department of Clinical Biochemistry, Army Medical University, Chongqing, 400038, China.
Abstract:
N6-methyladenosine (m6A), a dynamically reversible modification in eukaryotic RNAs, modulates gene expression and pathological processes in various tumors. KIAA1429, the largest component of the m6A methyltransferase complex, plays an important role in m6A modification. However, the underlying mechanism of KIAA1429 in hepatocellular carcinoma (HCC) remains largely unknown. Immunohistochemical assay was performed to examine the expression of KIAA1429 in HCC tissues. Transwell, wound healing and animal experiments were used to investigate the influence of KIAA1429 on cell migration and invasion. The mRNA high-throughput sequencing (RNA-seq) and methylated RNA immunoprecipitation sequencing (MeRIP-seq) were performed to screen the downstream target of KIAA1429. RNA stability assays, RNA immunoprecipitation assay (RIP), MeRIP-qPCR and luciferase assay were used to evaluate the relationship between KIAA1429 and the m6A-modified genes. Results showed that the expression level of KIAA1429 was significantly higher in HCC tissues than in adjacent tissues, and the upregulation of KIAA1429 could promote HCC metastasis in vitro and in vivo. Mechanistically, we confirmed that KIAA1429 negatively regulated the tumor suppressor, Rho family GTPase 3 (RND3), by decreasing its mRNA stability in coordination with the m6A reader YTHDC1. Moreover, we demonstrated that KIAA1429 could regulate the m6A modification of RND3 mRNA via its RNA binding domain. Our data indicated that KIAA1429 exerted its oncogenic role by inhibiting RND3 expression in an m6A-dependent manner, suggesting that KIAA1429 might be a potential prognostic biomarker and therapeutic target in HCC.
Insights
KIAA1429 promotes hepatocellular carcinoma (HCC) metastasis by reducing the tumor suppressor RND3 via m6A modification. This suggests KIAA1429 as a potential biomarker and therapeutic target for HCC.
Area of Science:
- Molecular Biology
- Oncology
- Epigenetics
Background:
- N6-methyladenosine (m6A) is a reversible RNA modification influencing gene expression and cancer.
- KIAA1429 is a key component of the m6A methyltransferase complex, but its role in hepatocellular carcinoma (HCC) is unclear.
Purpose of the Study:
- To investigate the mechanism of KIAA1429 in HCC progression.
- To identify downstream targets and regulatory pathways of KIAA1429 in HCC.
Main Methods:
- Immunohistochemistry to assess KIAA1429 expression in HCC tissues.
- In vitro (Transwell, wound healing) and in vivo experiments to evaluate HCC cell migration and invasion.
- RNA sequencing (RNA-seq) and methylated RNA immunoprecipitation sequencing (MeRIP-seq) to identify KIAA1429 targets.
- RNA stability assays, RIP, MeRIP-qPCR, and luciferase assays to confirm molecular mechanisms.
Main Results:
- KIAA1429 expression is significantly upregulated in HCC tissues and promotes HCC metastasis.
- KIAA1429 negatively regulates the tumor suppressor RND3 by decreasing its mRNA stability through m6A modification, in conjunction with YTHDC1.
- KIAA1429 directly binds and modifies RND3 mRNA via its RNA binding domain.
Conclusions:
- KIAA1429 promotes HCC oncogenesis by inhibiting RND3 expression in an m6A-dependent manner.
- KIAA1429 represents a potential prognostic biomarker and therapeutic target for hepatocellular carcinoma.
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