KIAA1429 facilitates metastasis via m6A-YTHDC1-dependent RND3 down-regulation in hepatocellular carcinoma cells

Meihua Shan1, Dong Liu1, Liangbo Sun1

  • 1Department of Clinical Biochemistry, Army Medical University, Chongqing, 400038, China.

Cancer Letters
|January 15, 2024
PubMed

Insights

KIAA1429 promotes hepatocellular carcinoma (HCC) metastasis by reducing the tumor suppressor RND3 via m6A modification. This suggests KIAA1429 as a potential biomarker and therapeutic target for HCC.

Area of Science:

  • Molecular Biology
  • Oncology
  • Epigenetics

Background:

  • N6-methyladenosine (m6A) is a reversible RNA modification influencing gene expression and cancer.
  • KIAA1429 is a key component of the m6A methyltransferase complex, but its role in hepatocellular carcinoma (HCC) is unclear.

Purpose of the Study:

  • To investigate the mechanism of KIAA1429 in HCC progression.
  • To identify downstream targets and regulatory pathways of KIAA1429 in HCC.

Main Methods:

  • Immunohistochemistry to assess KIAA1429 expression in HCC tissues.
  • In vitro (Transwell, wound healing) and in vivo experiments to evaluate HCC cell migration and invasion.
  • RNA sequencing (RNA-seq) and methylated RNA immunoprecipitation sequencing (MeRIP-seq) to identify KIAA1429 targets.
  • RNA stability assays, RIP, MeRIP-qPCR, and luciferase assays to confirm molecular mechanisms.

Main Results:

  • KIAA1429 expression is significantly upregulated in HCC tissues and promotes HCC metastasis.
  • KIAA1429 negatively regulates the tumor suppressor RND3 by decreasing its mRNA stability through m6A modification, in conjunction with YTHDC1.
  • KIAA1429 directly binds and modifies RND3 mRNA via its RNA binding domain.

Conclusions:

  • KIAA1429 promotes HCC oncogenesis by inhibiting RND3 expression in an m6A-dependent manner.
  • KIAA1429 represents a potential prognostic biomarker and therapeutic target for hepatocellular carcinoma.

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