Related Experiment Video

Updated: Aug 9, 2026

Mammary Epithelial Transplant Procedure
08:51

Mammary Epithelial Transplant Procedure

Published on: June 10, 2010

Toxic shock syndrome after augmentation mammaplasty

J Giesecke, C Arnander

    Annals of Plastic Surgery
    |December 1, 1986
    PubMed

    Insights

    Toxic shock syndrome (TSS) can occur from Staphylococcus aureus infections beyond tampon use. This case highlights TSS developing after augmentation mammaplasty surgery.

    Area of Science:

    • Medicine
    • Infectious Diseases
    • Surgery

    Background:

    • Toxic shock syndrome (TSS) was first described in 1978, initially observed in children.
    • A strong association with Staphylococcus aureus colonization of vaginal tampons was identified in 1980.
    • TSS can manifest from staphylococcal infections at various body sites, independent of foreign materials.

    Observation:

    • This report details a case of a patient who developed toxic shock syndrome following augmentation mammaplasty.
    • The patient's condition presented a clinical picture consistent with TSS.

    Findings:

    • The occurrence of TSS in this patient was linked to Staphylococcus aureus.
    • The absence of typical risk factors like tampon use suggests broader etiologies for TSS.

    Implications:

    • This case underscores that toxic shock syndrome can arise from staphylococcal infections associated with surgical procedures, such as augmentation mammaplasty.
    • It broadens the understanding of TSS etiology beyond its historically recognized associations.
    • Clinicians should consider TSS in patients presenting with compatible symptoms post-surgery.

    More Related Videos

    Endoscopic Bilateral Nipple-sparing Mastectomy via a Single Axillary Incision with Immediate Pre-pectoral Implant-based Breast Reconstruction13:35

    Endoscopic Bilateral Nipple-sparing Mastectomy via a Single Axillary Incision with Immediate Pre-pectoral Implant-based Breast Reconstruction

    Published on: May 17, 2024

    Single-Port Robotic-assisted Transaxillary Breast-conserving Surgery: A Prospective, Single-arm, Non-randomized Phase IIa Clinical Trial03:07

    Single-Port Robotic-assisted Transaxillary Breast-conserving Surgery: A Prospective, Single-arm, Non-randomized Phase IIa Clinical Trial

    Published on: August 19, 2025

    Related Concept Videos

    Aneurysm IV: Nursing Management01:22

    Aneurysm IV: Nursing Management

    Vigilant monitoring for aneurysm rupture is essential for patients undergoing aortic surgery.Preoperative Nursing ManagementContinuously monitor the patient for manifestations of aneurysm rupture, such as pallor, weakness, tachycardia, hypotension, abdominal, back, groin, or periumbilical pain, changes in consciousness, and a pulsating abdominal mass. Regularly assess the patient's peripheral pulses.Instruct the patient to consume a clear liquid diet the day before surgery and administer...
    Staphylococcal Skin Infections01:29

    Staphylococcal Skin Infections

    Staphylococcus aureus is a Gram-positive coccus that resides harmlessly on the skin and mucous membranes of healthy individuals. When the skin barrier is breached, it can shift from a commensal to an opportunistic pathogen. This transition is facilitated by surface adhesins, such as clumping factor B and S. aureus surface protein G (SasG), which bind to structural proteins, including loricrin and cytokeratin, in the damaged epidermis. Protein A, another key factor, binds the Fc region of...
    Healing II: Complications01:24

    Healing II: Complications

    Complications during healing arise when tissue repair is altered by local or systemic factors. These changes involve abnormal collagen deposition, altered biomechanics, and reduced vascular supply, impairing restoration of normal structure and function.Loss of FunctionScar tissue differs significantly from the original tissue it replaces. In the skin, fibrosis lacks adnexal structures such as hair follicles, sebaceous glands, and sweat glands. Their absence reduces tactile sensitivity, impairs...
    Cytotoxic Edema: Pathophysiology01:21

    Cytotoxic Edema: Pathophysiology

    Cytotoxic edema is a form of cerebral edema characterized by intracellular swelling of neurons, astrocytes, and other glial cells. It develops when the mechanisms responsible for maintaining ionic gradients across the cell membrane become impaired. Under normal physiological conditions, the sodium–potassium ATPase actively transports sodium ions out of the cell and potassium ions into the cell, preserving osmotic balance and enabling electrical signaling. This pump requires a continuous supply...