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Updated: Jun 17, 2026

Controlled Cortical Impact Model for Traumatic Brain Injury
Published on: August 5, 2014
Catatonia as a Result of a Traumatic Brain Injury
Jessica Berthelot1, Jacob Cambre2, Madeline Erwin2
1Department of Psychiatry, LSU School of Medicine, 5246 Brittany Drive, 3rd floor, Baton Rouge 70808, LA, USA.
Abstract:
Catatonia is a neuropsychiatric syndrome typically marked by disturbances in motor activity, speech, and behavior. It has historically been associated with psychiatric illness, but acute medical illness, neurocognitive disorders, and neurodevelopmental disorders can cause catatonia as well. Catatonia is likely underrecognized and underdiagnosed in the general medical hospital, despite high risks of morbidity and mortality and the availability of rapidly effective treatment. Here, we present a case of catatonia secondary to traumatic brain injury that responded to lorazepam after a delayed diagnosis. A young male patient who was incarcerated and assaulted was sent to the emergency department multiple times for unresponsive and unpredictable behavior, including not agreeing to be released home. After being admitted with the diagnosis of postconcussive syndrome, he was ultimately diagnosed with catatonia, and intravenous lorazepam resulted in a return to his baseline mental status. We discuss factors that led to the delay in diagnosis, including lack of training in recognition of catatonia, suspicion of feigned symptoms for secondary gain, and the implication of stigma in an African American young male arrested for a drug-related crime.
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