Early life phenobarbital exposure dysregulates the hippocampal transcriptome
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Updated: May 4, 2026

Analysis of Gene Expression Changes in the Rat Hippocampus After Deep Brain Stimulation of the Anterior Thalamic Nucleus
Published on: March 8, 2015
Seán Quinlan1, Tahiyana Khan1,2, David McFall1,2
1Department of Physiology and Pharmacology, Georgetown University, Washington, DC, United States.
Insights
Phenobarbital (PB) causes broad gene expression changes in neonatal brains, unlike levetiracetam (LEV). This transcriptomic alteration in developing brains may explain PB
Area of Science:
- Neuroscience
- Developmental Biology
- Pharmacology
Background:
- Phenobarbital (PB) and levetiracetam (LEV) are first-line neonatal seizure treatments.
- Concerns exist regarding PB's potential for lasting neurodevelopmental toxicity.
- The impact of these drugs on the neonatal brain's transcriptomic landscape is not fully understood.
Purpose of the Study:
- To investigate the effects of PB and LEV on the neonatal rat hippocampal transcriptome.
- To compare the transcriptomic changes induced by PB and LEV exposure during critical neurodevelopmental windows.
Main Methods:
- Neonatal rat pups received a single dose of PB, LEV, or vehicle.
- Hippocampal RNA sequencing was performed 72 hours post-administration.
- Differential gene expression analysis was conducted to identify transcriptomic alterations.
Main Results:
- PB exposure resulted in 124 differentially expressed transcripts, while LEV exposure affected only 15 transcripts.
- PB significantly decreased GABAergic and oligodendrocyte markers (pvalb, opalin).
- PB exposure increased markers of microglial activation (cd68) and astrocyte activity (vegfa).
Conclusions:
- PB induces widespread transcriptomic changes in the developing brain, suggesting a link to neurotoxicity.
- LEV appears to have a minimal impact on the neonatal transcriptome compared to PB.
- These findings highlight PB's potential to disrupt multiple cell types and molecular pathways during critical neurodevelopmental periods.
Abstract:
Introduction: Phenobarbital (PB) and levetiracetam (LEV) are the first-line therapies for neonates with diagnosed seizures, however, a growing body of evidence shows that these drugs given during critical developmental windows trigger lasting molecular changes in the brain. While the targets and mechanism of action of these drugs are well understood-what is not known is how these drugs alter the transcriptomic landscape, and therefore molecular profile/gene expression during these critical windows of neurodevelopment. PB is associated with a range of neurotoxic effects in developing animals, from cell death to altered synaptic development to lasting behavioral impairment. LEV does not produce these effects. Methods: Here we evaluated the effects of PB and Lev on the hippocampal transcriptome by RNA sequencing. Neonatal rat pups were given a single dose of PB, Lev or vehicle and sacrificed 72 h later-at time at which drug is expected to be cleared. Results: We found PB induces broad changes in the transcriptomic profile (124 differentially expressed transcripts), as compared to relatively small changes in LEV-treated animals (15 transcripts). PB exposure decreased GABAergic and oligodendrocyte markers pvalb and opalin, and increased the marker of activated microglia, cd68 and the astrocyte- associated gene vegfa. These data are consistent with the existing literature showing developmental neurotoxicity associated with PB, but not LEV. Discussion: The widespread change in gene expression after PB, which affected transcripts reflective of multiple cell types, may provide a link between acute drug administration and lasting drug toxicity.
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