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[Shock. A review (author's transl)].

C K Svendsen, R K Hjortkjaer

    Nordisk Veterinaermedicin
    |July 1, 1979
    PubMed
    Summary
    This summary is machine-generated.

    Shock, a critical circulatory inadequacy, arises from over 100 primary diseases. This summary explains shock pathogenesis, clinical signs, and emphasizes fluid volume expansion as the primary treatment for hypovolemic, vasogenic, and cardiogenic shock.

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    Area of Science:

    • Veterinary Medicine
    • Pathophysiology
    • Critical Care

    Context:

    • Shock is a life-threatening complication of numerous primary diseases.
    • It involves prolonged circulatory inadequacy, leading to insufficient tissue perfusion and cellular damage.
    • Understanding shock's etiology and pathogenesis is crucial for effective veterinary practice.

    Purpose:

    • To classify shock into hypovolemic, vasogenic, and cardiogenic types based on etiology.
    • To elucidate the pathogenesis of shock, using hypovolemic shock as a model.
    • To compare the pathogenesis of different shock types and highlight the central role of the sympatho-adrenal response.

    Summary:

    • Shock pathogenesis involves a sympatho-adrenal response leading to vasoconstriction and ischemia, progressing to irreversible stages without intervention.

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  • Key metabolic alterations include lacticacidemia and metabolic acidosis.
  • Clinical signs such as increased heart rate, altered mucosal perfusion, and hypothermia are directly linked to the sympatho-adrenal response.
  • Morphological changes include organ edema, hemorrhage, thrombosis, and degenerative alterations.

    • Impact:

    • Provides a comprehensive overview of shock, aiding in diagnosis and treatment strategies in veterinary medicine.
    • Highlights the importance of recognizing clinical signs related to the sympatho-adrenal response.
    • Emphasizes fluid volume expansion with balanced electrolyte solutions as the cornerstone of shock therapy.