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Vision Training Methods for Sports Concussion Mitigation and Management
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Cuproptosis and physical training.

Negin Kordi1, Ali Saydi1, Maliheh Azimi2

  • 1Department of Exercise Physiology, Faculty of Sport Sciences, Razi University, Kermanshah, Iran.

Clinical Hemorheology and Microcirculation
|July 20, 2024
PubMed
Summary

Cuproptosis, a novel cell death pathway, is linked to cardiovascular diseases. Physical training may inhibit cuproptosis by reducing oxidative stress, offering a potential therapeutic strategy.

Keywords:
Coppercell deathcuproptosisexercisephysical activity

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Area of Science:

  • Cellular Biology
  • Metabolic Pathways
  • Toxicology

Background:

  • Copper is vital for human health, regulating key metabolic functions.
  • Disruptions in copper homeostasis are implicated in various diseases, including cardiovascular conditions.
  • Cuproptosis, a distinct cell death mechanism, is dependent on copper and mitochondrial respiration.

Purpose of the Study:

  • To explore the role of cuproptosis in cardiovascular disease.
  • To investigate potential therapeutic interventions for cuproptosis-related conditions.

Main Methods:

  • Review of existing literature on copper homeostasis, cuproptosis, and cardiovascular disease.
  • Analysis of molecular mechanisms underlying copper-induced cell death.
  • Exploration of the potential of physical training as an inhibitor of cuproptosis.

Main Results:

  • Excess copper induces cuproptosis through protein toxic stress and iron-sulfur cluster degradation.
  • Copper transporters play a role in regulating cuproptosis.
  • Copper-chelating agents and physical training show potential in mitigating cuproptosis.

Conclusions:

  • Cuproptosis is implicated in cardiovascular disease pathogenesis.
  • Physical training may serve as a strategy to inhibit cuproptosis by targeting protein stress responses.
  • Further research into cuproptosis inhibitors is warranted for cardiovascular disease management.