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Relationship between age and cellular suppressive activity in resistance to Histoplasma capsulatum infection

Sabouraudia
|October 1, 1985
PubMed

Insights

Young BALB/c mice spleen cells transfer suppressed adult mice resistance to Histoplasma capsulatum infection. This indicates a transferable suppressive state mediated by specific cell populations.

Area of Science:

  • Immunology
  • Infectious Diseases
  • Microbiology

Background:

  • Age significantly impacts BALB/c mice resistance to Histoplasma capsulatum.
  • Younger mice (1-month-old) exhibit lower resistance compared to adult mice (4.5-month-old).

Purpose of the Study:

  • To investigate the immunological mechanisms underlying age-dependent resistance to Histoplasma capsulatum.
  • To determine if the reduced resistance in young mice is transferable to adult mice.

Main Methods:

  • Comparative infection studies using BALB/c mice of different ages (1-month, 4.5-month, and 1-year-old).
  • Adoptive transfer of spleen cells from young (1-month-old) to adult (4.5-month-old) syngeneic mice.
  • Infection challenge with varying doses (LD50, LD100) of Histoplasma capsulatum.
  • Evaluation of survival rates and clinical course in recipient mice.

Main Results:

  • 4.5-month-old mice normally resolved Histoplasma capsulatum infection.
  • Adoptive transfer of spleen cells from 1-month-old mice to 4.5-month-old mice resulted in a critical clinical course and mortality.
  • Irradiated mice receiving bone marrow and spleen cells from young mice succumbed to infection.
  • Non-irradiated adult mice receiving only spleen cells from young mice also showed suppressed resistance.
  • The suppressive state appears to require both non-adherent and adherent, radioresistant spleen cell populations from young mice.

Conclusions:

  • Spleen cells from young mice transfer a suppressive state that abrogates the natural resistance of adult mice to Histoplasma capsulatum.
  • This age-related immune suppression is mediated by specific cell populations within the spleen.
  • Findings suggest a complex interplay of cellular immunity and age in controlling fungal infections.

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