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Testosterone and estradiol reduce inflammation of human macrophages induced by anti-SARS-CoV-2 IgG
Sona Allahverdiyeva1,2, Chiara E Geyer1, Jennifer Veth1
1Center for Experimental and Molecular Medicine, Amsterdam Institute for Infection and Immunity, Amsterdam University Medical Center, Amsterdam, the Netherlands.
Abstract:
COVID-19, the disease caused by SARS-CoV-2, particularly causes severe inflammatory disease in elderly, obese, and male patients. Since both aging and obesity are associated with decreased testosterone and estradiol expression, we hypothesized that decreased hormone levels contribute to excessive inflammation in the context of COVID-19. Previously, we and others have shown that hyperinflammation in severe COVID-19 patients is induced by the production of pathogenic anti-spike IgG antibodies that activate alveolar macrophages. Therefore, we developed an in vitro assay in which we stimulated human macrophages with viral stimuli, anti-spike IgG immune complexes, and different sex hormones. Treatment with levels of testosterone reflecting young adults led to a significant reduction in TNF and IFN-γ production by human macrophages. In addition, estradiol significantly attenuated the production of a very broad panel of cytokines, including TNF, IL-1β, IL-6, IL-10, and IFN-γ. Both testosterone and estradiol reduced the expression of Fc gamma receptors IIa and III, the two main receptors responsible for anti-spike IgG-induced inflammation. Combined, these findings indicate that sex hormones reduce the inflammatory response of human alveolar macrophages to specific COVID-19-associated stimuli, thereby providing a potential immunological mechanism for the development of severe COVID-19 in both older male and female patients.
Insights
Sex hormones like testosterone and estradiol may protect against severe COVID-19 by reducing inflammation. Lower hormone levels in older or obese individuals could explain increased disease severity and hyperinflammation.
Area of Science:
- Immunology
- Endocrinology
- Virology
Background:
- COVID-19 severity is linked to age, obesity, and male sex.
- Decreased sex hormone levels (testosterone, estradiol) correlate with aging and obesity.
- Hyperinflammation in severe COVID-19 involves anti-spike IgG antibodies activating macrophages.
Purpose of the Study:
- To investigate the role of sex hormones in modulating COVID-19-induced inflammation.
- To determine if testosterone and estradiol can reduce inflammatory responses in human macrophages.
Main Methods:
- Developed an in vitro assay stimulating human macrophages.
- Macrophages were exposed to viral stimuli, anti-spike IgG immune complexes, and sex hormones.
- Measured cytokine production (TNF, IFN-γ, IL-1β, IL-6, IL-10) and Fc gamma receptor expression.
Main Results:
- Testosterone significantly reduced TNF and IFN-γ production.
- Estradiol attenuated a broad panel of cytokines, including TNF, IL-1β, IL-6, and IFN-γ.
- Both hormones decreased Fc gamma receptors IIa and III expression on macrophages.
Conclusions:
- Sex hormones mitigate inflammatory responses in human macrophages against COVID-19 stimuli.
- Reduced sex hormone levels may contribute to severe COVID-19 and hyperinflammation in vulnerable populations.
- This provides a potential immunological mechanism for sex and age disparities in COVID-19 outcomes.

