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The role of feeding rejection in Drosophila mutation assays

Mutation Research
|September 1, 1985
PubMed

Insights

5-bromo-2-deoxyuridine (BUdR) reduces mutagen uptake in Drosophila, lowering mutation rates. This highlights the importance of monitoring feeding behavior in mutagenicity studies.

Area of Science:

  • Genetics
  • Toxicology
  • Drosophila melanogaster research

Background:

  • Sex-linked recessive lethal mutations are studied to assess mutagenicity.
  • 5-bromo-2-deoxyuridine (BUdR) is a thymidine analog that can be incorporated into DNA.
  • Previous studies suggested potential interactions between BUdR and mutagens.

Purpose of the Study:

  • To investigate the effect of BUdR on the frequency of sex-linked recessive lethal mutations induced by methyl methanesulfonate (MMS) and ethyl methanesulfonate (EMS) in Drosophila.
  • To determine if BUdR exhibits a protective effect against mutagen-induced mutations.
  • To elucidate the mechanism behind any observed protective effect, focusing on mutagen uptake.

Main Methods:

  • Drosophila melanogaster males were fed solutions containing MMS or EMS, with or without BUdR.
  • Mutation frequencies were assessed by scoring sex-linked recessive lethal mutations in post-meiotic germ cells.
  • [14C]sucrose was used to quantify the uptake of feeding solutions by the flies.
  • Feeding behavior and solution uptake were compared between treatment groups.

Main Results:

  • The presence of BUdR significantly reduced the frequency of induced mutations compared to mutagens alone.
  • BUdR significantly inhibited the uptake of the feeding solution containing mutagens.
  • Reduced mutagen uptake due to BUdR can explain the observed decrease in mutation rates.

Conclusions:

  • BUdR appears to reduce mutagenicity not by direct chemical interaction, but by decreasing the flies' intake of the mutagen.
  • These findings underscore the critical need to assess and control for feeding behavior in adult feeding studies involving mutagens.
  • The study provides a mechanistic explanation for the apparent protective effect of BUdR in mutagenicity assays.

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