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Lethal effect of male rat submandibular gland homogenate for rat neonates

Journal of Oral Pathology
|September 1, 1985
PubMed

Insights

Suckling rats injected with male submandibular gland homogenate (SMGH) died due to absorbed trypsin-like enzymes. Adult rats tolerated SMGH, suggesting age-dependent enzyme inactivation is critical for survival.

Area of Science:

  • Toxicology
  • Biochemistry
  • Developmental Biology

Background:

  • The submandibular gland (SMG) produces various bioactive compounds, including enzymes.
  • The physiological impact of SMG homogenate, particularly in developing organisms, is not fully understood.

Purpose of the Study:

  • To investigate the toxicity of male submandibular gland homogenate (SMGH) in rats of different ages.
  • To determine the role of age-dependent factors in the response to SMGH exposure.

Main Methods:

  • Subcutaneous injection of male SMGH and liver homogenate (LH) into suckling, weanling, and adult rats.
  • Monitoring survival rates within 24 hours post-injection.
  • Quantifying serum trypsin-like activity in injected rats.

Main Results:

  • Suckling rats injected with SMGH exhibited high mortality within 24 hours, unlike those receiving LH or older rats receiving SMGH.
  • A significant increase (1200%) in serum trypsin-like activity was observed in surviving suckling rats after SMGH injection.
  • Adult rats showed a much smaller increase (46%) in serum trypsin-like activity following a proportionally similar SMGH dose.

Conclusions:

  • Suckling rats are highly susceptible to the toxic effects of male SMGH, likely due to impaired enzyme inactivation.
  • The observed mortality in pups is attributed to the absorption of active trypsin-like enzymes leading to a hypotensive crisis.
  • Age-dependent differences in enzyme metabolism play a crucial role in determining the toxicity of SMGH.

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